ArticleTAG. Theoretical and applied genetics. Theoretische und angewandte Genetik2025
Genetic loci and novel candidate causal genes for preharvest sprouting resistance in wheat (Triticum aestivum L.).
Article in TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
key messageGWAS and targeted transcriptomic analysis identified three QTLs and novel candidate causal genes for preharvest sprouting resistance in wheat. Preharvest sprouting (PHS), which causes significant yield and quality losses in wheat, is primarily associated with low level of seed dormancy. This study conducted a genome-wide association analysis of a diverse panel of spring wheat genotypes across five environments to identify loci and candidate causal genes controlling seed dormancy/PHS resistance. Phenotypic evaluation of the mapping panel revealed substantial variation in seed dormancy levels/PHS resistance among the genotypes. Marker-trait association analysis identified 14 SNPs significantly linked to seed dormancy/PHS resistance based on a false discovery rate (FDR) of α = 0.05. These SNPs are mapped to three QTLs on chromosomes 4A, 5B, and 5D. The QTL on 4A, QPhs.umb-4A, which consists of 11 SNPs that explained 10.7 to 20.8% of the phenotypic variation, is the most significant locus. The QTLs on 5B (QPhs.umb-5B) and 5D (QPhs.umb-5D) accounted for 10.2 to 11.4% of the phenotypic variation. A total of 730 unique candidate genes are associated with the three QTLs, and targeted transcriptomic analysis of selected candidate genes offered insights into their potential role in controlling seed dormancy and PHS resistance. The identified SNPs and candidate genes have the potential to enhance marker-assisted selection for improved PHS resistance in wheat.
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