Evidence map›Paper›PMID 40600019›Full record

ReviewFrontiers in endocrinology2025

Neuroendocrine neoplasms as a lynch syndrome manifestation: a case report and comprehensive literature review.

Maria Paula Bernal Zárate, Daniel Felipe Mendivelso-Gonzalez, William Camilo Torres, Angelica Maria González Clavijo, Diego Felipe Ballen, Rafael Parra Medina, Julián C Riaño-Moreno

Abstract readCase ReportsReview
In one paragraph

Review in Frontiers in endocrinology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Review
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Maria Paula Bernal ZárateDepartment of Genetics, Fundación Universitaria de Ciencias de la Salud, Bogotá, Colombia.
Daniel Felipe Mendivelso-GonzalezDepartment of Pathology and Molecular Oncology, Instituto Nacional de Cancerología, Bogotá, D.C., Colombia.
William Camilo TorresDepartment of Pathology and Molecular Oncology, Instituto Nacional de Cancerología, Bogotá, D.C., Colombia.
Angelica Maria González ClavijoDepartment of Endocrinology, Instituto Nacional de Cancerología, Bogotá, D.C., Colombia.
Diego Felipe BallenDepartment of Endocrinology, Instituto Nacional de Cancerología, Bogotá, D.C., Colombia.
Rafael Parra MedinaDepartment of Pathology and Molecular Oncology, Instituto Nacional de Cancerología, Bogotá, D.C., Colombia.
Julián C Riaño-MorenoDepartment of Pathology and Molecular Oncology, Instituto Nacional de Cancerología, Bogotá, D.C., Colombia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Lynch syndrome (LS) is an autosomal dominant inherited disorder caused by pathogenic variants in DNA mismatch repair (MMR) genes, most commonly

Indexed as

Colorectal Neoplasms, Hereditary NonpolyposisNeuroendocrine TumorsFemaleHumansMiddle AgedMutL Protein Homolog 1MLH1 protein, humanMutL Protein Homolog 1hereditary cancerlynch syndromemismatch repair-deficient (dMMR)neuroendocrine neoplasms (NENs)neuroendocrine tumor (NET)

Identifiers

PMID40600019
PMCPMC12208827

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.