ArticleCJC open2025
Microcirculation Function in Non-ST-Elevation Myocardial Infarction After the Index Event and at Follow-Up Assessment.
Article in CJC open, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Prognostic Impact of Infarct Size and Coronary Microvascular Function Via Cardiovascular Magnetic Resonance and Coronary Angiography-Derived Index of Microcirculatory Resistance in Myocardial Infarction With Nonobstructive Coronary Arteries.Journal of the American Heart Association · 2026Observational
Corrections and comments
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: The assessment of coronary physiology is seldom considered in cases of non-ST-elevation myocardial infarction (NSTEMI). This study aimed to characterize coronary physiology and determine the incidence of microvascular dysfunction in the myocardial infarction (MI) culprit coronary artery during the acute phase of NSTEMI and subsequent follow-up evaluation. Methods: This study included 30 patients hospitalized for NSTEMI. A physiological assessment of the MI culprit coronary artery was performed using fractional flow reserve, coronary flow reserve (CFR), and index of microcirculatory resistance (IMR). At a median of 7 months after MI, patients underwent repeated physiological assessment of the same coronary artery. Results: Microvascular dysfunction identified using CFR (< 2.0) was frequently present (60%) during the acute phase of NSTEMI, whereas severe microcirculatory dysfunction (IMR > 40) was uncommon (17%). Over time, a significant reduction occurred in the prevalence of abnormal CFR values (< 2.0; 60% vs 26%, Conclusions: In the acute phase of NSTEMI, reduced CFR was commonly observed in the MI culprit coronary artery, whereas severe microcirculatory dysfunction was infrequent.
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Registered trials
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