ArticleParasitology research2025
First complete mitochondrial genome of Armillifer moniliformis (Pentastomida: Porocephalida) isolated from a human case in Northern Thailand: comparative and phylogenetic analyses.
Article in Parasitology research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
2 citing papers in PubMed.
- Complete mitochondrial genome pathological characteristics and scanning electron microscopic observations ofInternational journal for parasitology. Parasites and wildlife · 2026Article
- Current status and prospects of nanopore sequencing technology in the detection of pathogenic microorganisms.Frontiers in microbiology · 2026Review
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Authors and funding
6 authors.
Funding
Abstract
Pentastomiasis, a zoonotic parasitic disease caused by pentastomids, has been increasingly documented in Thailand. Historically, morphological identification has been the only diagnostic method, but it is inadequate due to morphological similarities between larval stages and limited taxonomic keys. To overcome this, molecular diagnosis is required. However, the DNA sequence information of these neglected parasites remains largely unknown. In this study, pentastomid larvae were collected from the greater omentum of a patient in Northern Thailand. The species of pentastomid larvae was molecularly identified as Armillifer moniliformis, using PCRs targeting nuclear and mitochondrial regions, followed by Sanger sequencing. Nanopore sequencing was employed to characterize the complete mitochondrial genome of A. moniliformis, revealing a total length of 16,367 bp with 37 mitochondrial genes, including 13 protein-coding genes, 22 tRNA genes, and two rRNA genes, as well as one non-coding region. The entire mitogenome is highly AT-rich (62.1%), with nucleotide skews indicating compositional bias between strands. The non-coding region (2616 bp) lies between the tRNA-Leu
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