ArticleMolecular diversity2026
In silico prediction of drug-induced cardiotoxicity with ensemble machine learning and structural pattern recognition.
Article in Molecular diversity, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
4 citing papers in PubMed.
- A benchmark dataset and interpretable deep learning framework for drug-induced developmental neurotoxicity prediction.RSC advances · 2026Article
- Targeting IDH-driven Oncometabolism in glioblastoma: a computational approach for the identification of potential inhibitors.Frontiers in bioinformatics · 2026Article
- Review
- Analysis of the Toxicological Profile ofPlants (Basel, Switzerland) · 2025Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
6 authors.
Funding
Abstract
Drug-induced cardiotoxicity poses a significant risk to human health, and reliable predictive models are needed for safety assessment. In this study, a range of machine and deep learning models were developed for five cardiotoxicity end points, including heart failure (HF), arrhythmia (ARR), heart block (HB), hypertension (HP), and heart attack (HA). A total of 110 predictive models were constructed for each cardiotoxicity endpoint using various algorithms and molecular descriptors, and consensus models were developed based on the best-performing individual classifiers. The consensus models consistently outperformed individual models in cross-validation and external validation. Further molecular property analysis revealed that cardiotoxic compounds tend to exhibit higher molecular weight, increased lipophilicity (logP), lower hydrogen bonding capacity (HBD and HBA), and reduced topological polar surface area (TPSA). Additionally, key structural alerts (SAs), including secondary amines, benzene derivatives, sulfonamide/sulfonylurea groups, and heterocyclic structures, were identified. These SAs may mediate cardiotoxicity through ion channel inhibition, oxidative stress induction, and calcium homeostasis disruption. This study provides an integrated machine learning and deep learning computational framework for drug cardiotoxicity assessment and provides an exploration of the structural characteristics of cardiotoxic compounds, which is helpful for the discovery of safer drugs and chemical risk assessment.
Indexed as
Identifiers
40569519What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.