Article in mSystems, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
9 authors.
Sarunporn TandhavanantDepartment of Bacteriology, Institute of Tropical Medicine, Nagasaki University, Nagasaki, Japan.ORCID 0000-0003-0261-3233
Hiroyuki TerashimaDepartment of Bacteriology, Institute of Tropical Medicine, Nagasaki University, Nagasaki, Japan.
Hirotaka HiyoshiDepartment of Bacteriology, Institute of Tropical Medicine, Nagasaki University, Nagasaki, Japan.
Dhira Saraswati AnggramuktiDepartment of Bacterial Infections, Research Institute for Microbial Diseases, Osaka University, Osaka, Japan.
Nopadol PrechaDepartment of Microbiology and Immunology, Faculty of Tropical Medicine, Mahidol University, Bangkok, Thailand.
Tetsuya IidaDepartment of Bacterial Infections, Research Institute for Microbial Diseases, Osaka University, Osaka, Japan.ORCID 0000-0001-5134-1794
Shigeaki MatsudaDepartment of Bacterial Infections, Research Institute for Microbial Diseases, Osaka University, Osaka, Japan.ORCID 0000-0001-8430-9723
Narisara ChantratitaDepartment of Microbiology and Immunology, Faculty of Tropical Medicine, Mahidol University, Bangkok, Thailand.ORCID 0000-0003-3906-7379
Toshio KodamaDepartment of Bacteriology, Institute of Tropical Medicine, Nagasaki University, Nagasaki, Japan.ORCID 0000-0002-6511-7301
Funding
Faculty of Tropical Medicine, Mahidol UniversityJapan Agency for Medical Research and Development JP223fa627004Japan Agency for Medical Research and Development JP23fk0108663Japan Agency for Medical Research and Development JP23wm0125006Japan Agency for Medical Research and Development JP23wm0225023Japan Society for the Promotion of Science 21K07022Research Institute for Microbial Diseases, Osaka University JRPRIMD25B4
6 · The paper itself
Abstract
Many pathogenic bacteria regulate gene expression in response to the surrounding environment to establish infection. One such mechanism is the regulation of gene expression in response to contact with host cells. Here, we show that
Indexed as
Gene Expression Regulation, BacterialType III Secretion SystemsVibrio parahaemolyticusBacterial ProteinsGenomic IslandsHost-Pathogen InteractionsHumansVibrio InfectionsVirulenceVirulence FactorsBacterial ProteinsType III Secretion SystemsVirulence Factorsgatekeepergene regulationtype III secretion systemVibrio parahaemolyticus
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Regulatory mechanism for host-cell contact-dependent T3SS gene expression in · full record | OpenQuestion