Evidence map›Paper›PMID 40524452›Full record

ArticleBrain pathology (Zurich, Switzerland)2025

Contribution of α-synuclein cytopathologies to distinct seeding of misfolded α-synuclein.

Ain Kim, Ivan Martinez-Valbuena, Krisztina Danics, Shelley L Forrest, Gabor G Kovacs

Abstract read
In one paragraph

Article in Brain pathology (Zurich, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

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2 · The registry

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3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. From aggregate structure to disease trajectory: the role of the proteostasis network.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2026
    Review
  2. Article
4 · The record

Corrections and comments

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5 · Who and what money

Authors and funding

5 authors.

Ain KimDepartment of Laboratory Medicine & Pathobiology, University of Toronto, Toronto, Ontario, Canada.ORCID 0009-0005-3810-6390
Ivan Martinez-ValbuenaTanz Centre for Research in Neurodegenerative Disease, University of Toronto, Toronto, Ontario, Canada.ORCID 0000-0001-6463-6321
Krisztina DanicsDepartment of Forensic and Insurance Medicine, Semmelweis University, Budapest, Hungary.
Shelley L ForrestTanz Centre for Research in Neurodegenerative Disease, University of Toronto, Toronto, Ontario, Canada.ORCID 0000-0001-7863-5389
Gabor G KovacsDepartment of Laboratory Medicine & Pathobiology, University of Toronto, Toronto, Ontario, Canada.ORCID 0000-0003-3841-5511

Funding

Canadian Foundation for Innovation 40480Edmond J. Safra Philanthropic FoundationKrembil FoundationMaybank FoundationMultiple System Atrophy Coalition MSAC-2022-12-003NIH HHS 3200005870-24-112Rossy Family Foundation
6 · The paper itself

Abstract

Synucleinopathies are a group of neurodegenerative diseases characterized by the deposition of misfolded α-synuclein (αSyn), predominantly in oligodendrocytes in multiple system atrophy (MSA) and in neurons in Lewy body diseases (LBD). The contribution of αSyn cytopathologies to the pathogenesis of these diseases is underappreciated. Seed amplification assays of MSA and LBD brains have revealed striking differences in αSyn seeding between regions and cases. Therefore, our aim was to evaluate whether different brain regions containing distinct αSyn cytopathologies contribute to different seeding characteristics. We collected 2-mm micro-punches of regions in MSA (n = 10) and LBD (n = 15) cases from formalin-fixed paraffin-embedded tissues. We performed double immuno-labeling for disease-associated αSyn and cellular markers on tissue microarrays, evaluated co-deposition of other neurodegenerative disease-related proteins and, from the same micro-punched samples, we analyzed αSyn seeding. Based on these variables, machine learning algorithms were used to reduce dimensionality of the dataset and cluster the regions in MSA and LBD cases, revealing that different compositions of αSyn cytopathologies influence αSyn seeding patterns. Our results support the notion of different cellular processing of αSyn and its contribution to the variability in seeding. This has implications for understanding disease progression, interpretation of seed amplification assays, and opens avenues for the development of cell type-specific antibodies against αSyn.

Indexed as

alpha-SynucleinBrainLewy Body DiseaseMultiple System AtrophySynucleinopathiesAgedAged, 80 and overFemaleHumansMaleMiddle AgedNeuronsOligodendrogliaalpha-Synucleinalpha‐synucleincell‐specificmachine learningseeding behavior

Identifiers

PMID40524452
PMCPMC12488258

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.