ReviewFrontiers in psychology2025
Neurobiological basis of autism spectrum disorder: mini review.
Review in Frontiers in psychology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers, 1 of them a synthesis that pooled it.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
5 citing papers in PubMed, 1 synthesis or guideline pooled it.
- Zinc deficiency and zinc/copper ratio imbalance in autism spectrum disorder: a reanalysis of six multinational studies.Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine · 2026Pooled it
- Depression in Autism Spectrum Disorder: Neurobiological Convergence and Emerging Therapeutic Strategies.Biology · 2026Review
- Comprehensive Assessment of Druggable Targets in Cortical Neurons Reveals Biological Limits of Cell Type-Specific Neuropharmacology.Biomedicines · 2026Article
- Analysis of NDNF and SLC1A2 gene expression in the dorsolateral prefrontal cortex of individuals with autism.Frontiers in neuroscience · 2026Article
- Targeted Endogenous Bioelectric Modulation in Autism Spectrum Disorder: Real-World Clinical Outcomes of the REAC BWO Neurodevelopment-Autism Protocol.Journal of clinical medicine · 2025Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
11 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Autism Spectrum Disorder is a neuropsychiatric condition characterized by deficits in communication, social interaction, and repetitive behaviors, with significant symptom variability. This multifaceted profile reflects a complex genetic architecture as well as diversity in morphological characteristics. Therefore, the objective of this review is to discuss the genetic and morphological aspects that may contribute to understanding autism. No temporal restrictions were applied for study inclusion, nor were there limitations regarding language. Scientific articles available in full text and directly related to the topic were included, while editorials, letters, conference abstracts, theses, dissertations, and books were excluded. The results of this review converge on two main aspects: (1) genetic and morphological findings are essential for a more comprehensive understanding of the disorder, providing an important basis for investigating its underlying mechanisms; and (2) despite their relevance, the results are still incipient and insufficient to explain the full clinical and behavioral heterogeneity associated with autism, highlighting the need for further studies.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.