ArticleIndian journal of orthopaedics2025
Basic Science Research in Frozen Shoulder: Current Updates.
Article in Indian journal of orthopaedics, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
4 citing papers in PubMed.
- Progress in the Treatment of Adhesive Capsulitis with External Therapies of Traditional Chinese Medicine: A Comprehensive Review.Journal of pain research · 2026Review
- Signaling pathway networks in frozen shoulder fibrosis: from inflammatory initiation to fibrotic progression.Frontiers in medicine · 2026Review
- Frozen shoulder: a narrative review of current treatment concepts and the underlying scientific evidence.Clinics in shoulder and elbow · 2025Article
- Fibroblast growth factor 9 activates fibroblast activation and drives the progress of shoulder stiffness.Frontiers in cell and developmental biology · 2025Article
Corrections and comments
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Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: Frozen shoulder (FS) is a painful and functionally debilitating condition characterized by progressive shoulder stiffness. The etiology and pathomechanism of FS remain unclear but are likely influenced by genetic predisposition, systemic inflammation, and localized shoulder trauma. Methods: A comprehensive search was conducted on July 4, 2024, using PubMed, SCOPUS, and Google Scholar. Keywords included "frozen shoulder" and "adhesive capsulitis," with a focus on basic science-related topics. Results: Chronic inflammatory conditions in FS trigger a cascade involving cytokines, growth factors, increased extracellular matrix turnover, fibroblast activation, collagen deposition, and reduced matrix degradation, contributing to fibrosis and impaired joint mobility. Conclusions: FS is a condition with multifactorial triggering factors that require targeted research to elucidate its mechanisms and identify molecular targets. Basic scientific studies are essential for developing biomarkers and advancing therapeutic strategies.
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Registered trials
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