Evidence map›Paper›PMID 40505974›Full record

ArticleBehavioural brain research2025

Evaluating alpha-synuclein proteinopathy and consequences for birdsong in zebra finch basal ganglia area X.

Reed T Bjork, Famesh Z Patel, Madeleine S Daly, Julie E Miller

Abstract read
In one paragraph

Article in Behavioural brain research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Reed T BjorkDepartment of Neuroscience, University of Arizona, Tucson, AZ, United States; Graduate Interdisciplinary Program in Neuroscience, University of Arizona, Tucson, AZ, United States.
Famesh Z PatelDepartment of Neuroscience, University of Arizona, Tucson, AZ, United States.
Madeleine S DalyDepartment of Neuroscience, University of Arizona, Tucson, AZ, United States.
Julie E MillerDepartment of Neuroscience, University of Arizona, Tucson, AZ, United States; Graduate Interdisciplinary Program in Neuroscience, University of Arizona, Tucson, AZ, United States; Speech, Language and Hearing Sciences, Neurology, BIO5 Institute, University of Arizona, Tucson, AZ, United States. Electronic address: juliemiller@arizona.edu.

Funding

Alpha-synuclein driven cellular changes and vocal dysfunction in Parkinson's DiseaseR21NS123512 · NINDS · UNIVERSITY OF ARIZONA · PI MILLER, JULIE ELIZABETH · 2021 to 2021
$415k
NINDS NIH HHS R21 NS123512
6 · The paper itself

Abstract

Lewy body pathology is a major hallmark of Parkinson's Disease (PD) and other dementias. The process of Lewy body formation is largely driven by the aggregation of alpha-synuclein (αsyn), an abundant presynaptic chaperone protein that has been shown to propagate between neurons when misfolded. Preclinical animal models inducing αsyn aggregation in the brain have demonstrated a range of behavioral consequences, though studies connecting behavioral changes to specific features of pathology are lacking. Considering vocal impairment manifests early in individuals with PD and fails to resolve upon dopamine replacement, we examined the effect of αsyn proteinopathy on birdsong in adult male zebra finches to investigate these early mechanisms of PD-related vocal dysfunction. In this study, we describe a novel tool for measuring αsyn expression called the Border Expression Ratio (BER) based on the discrete physiological distribution of αsyn surrounding basal ganglia song center, Area X. Following overexpression of human αsyn in Area X using bilateral injections of adeno-associated virus, we show that BER can be used to measure regional αsyn proteinopathy, revealing a positive correlation between right hemisphere pathology and a reduction in the variation of harmonic syllable duration. Finally, we provide evidence of serine 129 phosphorylation-a biomarker for aggregated αsyn-in Area X and cortical song nucleus LMAN of αsyn-overexpressing finches, despite this residue not being conserved in zebra finch αsyn, indicating modification of the human transgene. Together, these findings provide a framework for future analyses investigating αsyn propagation over time and the effects on vocal behavior.

Indexed as

alpha-SynucleinBasal GangliaSynucleinopathiesVocalization, AnimalAnimalsDisease Models, AnimalFinchesHumansMaleParkinson Diseasealpha-SynucleinAlpha-synucleinBasal gangliaBirdsongLewy bodyProteinopathyVocalZebra finch

Identifiers

PMID40505974
PMCPMC12276859

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.