Evidence map›Paper›PMID 40500501›Full record

ReviewAdvances in neurobiology2025

Neuroglial Pathophysiology of Leukodystrophies.

Alexei Verkhratsky, Jianqin Niu, Chenju Yi, Arthur Butt

Abstract readReview
PubMed Publisher
In one paragraph

Review in Advances in neurobiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Alexei VerkhratskyFaculty of Biology, Medicine and Health, The University of Manchester, Manchester, UK. Alexej.Verkhratsky@manchester.ac.uk.
Jianqin NiuDepartment of Histology and Embryology, Third Military Medical University, Chongqing, China.
Chenju YiResearch Centre, Seventh Affiliated Hospital of Sun Yat-sen University, Shenzhen, China.
Arthur ButtSchool of Medicine, Pharmacy and Biomedical Science, University of Portsmouth, Portsmouth, UK.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Leukodystrophies are a diverse group of inherited diseases characterised by white matter degenerative pathology. Leukodystrophies have a highly heterogeneous genetic background linked mainly to mutations in oligodendrocyte and astrocyte genes and, to lesser extent, microglia. The most prevalent leukodystrophies are caused by mutations in oligodendrocyte genes that encode the essential myelin proteins PLP1 and GalC in Pelizaeus-Merzbacher disease and Krabbe disease, respectively. Astrocyte leukodystrophies are led by Alexander disease, caused by mutations in the astrocyte gene GFAP. Vanishing white matter disease, the most prevalent inherited white matter pathology in children, is associated with astrocyte atrophy and cystic degeneration of the cerebral white matter. The pathogenic mechanisms in leukodystrophies depend on the genetic mutations and hence are extremely varied, but the diseases have in common white matter atrophy caused by the loss of oligodendrocytes and myelin, with or without marked reactive astrogliosis and microglia activation. The development of a range of animal models with the disruption of specific genes causing leukodystrophies and the use of pluripotent stem cells from people with different forms of leukodystrophy is advancing the understanding of the functional and cellular pathophysiology of these rare diseases.

Indexed as

AstrocytesLeukoencephalopathiesNeurogliaOligodendrogliaAnimalsHereditary Central Nervous System Demyelinating DiseasesHumansMicrogliaWhite MatterAicardi Goutières syndromeAlexander diseaseAstroglial leukodystrophiesCanavan diseaseKrabbe diseaseMerzbacher diseaseMicroglial leukodystrophiesNasu-Hakola diseaseOligodendroglial leukodystrophiesPelizaeusVanishing white matter disease

Identifiers

PMID40500501

What OpenQuestion holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.