ArticleACS nano2025
Silicon Carbide Nanowires Impair Mucociliary Clearance-Mediated Innate Immunity in Primary Human Bronchial Epithelial Cells.
Article in ACS nano, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Boron Nitride Nanomaterials Trigger Immunomodulatory Effects in Human Broncho-Epithelial Cells by Modulating Eicosanoid Lipid Signaling.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026Article
Corrections and comments
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Authors and funding
10 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The respiratory tract possesses mucociliary-driven innate immune defense mechanisms that protect the lungs from harmful environmental exposures, but when damaged, increase susceptibility to respiratory infections and diseases. Inhalation exposure to certain nanomaterials has been shown to trigger fibrosis and other respiratory conditions. However, there is a limited understanding of whether nanomaterials can impair mucociliary defense in lungs and its underlying mechanism. Here, we first investigated the fate of zero-dimensional, one-dimensional, and two-dimensional silicon- and carbon-based nanomaterials (silicon carbide nanowires (SiC NWs), silicon dioxide (SiO
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Registered trials
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