ReviewMolecular horticulture2025
The triggering mechanism for predominant hormonal signal production in fleshy fruit ripening.
Review in Molecular horticulture, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
4 citing papers in PubMed.
- Immature fruit abscission is triggered by hormonal changes in the seed coat of avocado.Journal of experimental botany · 2026Article
- SlSAM3 interacts with SlACS4 to facilitate brassinosteroid signaling-mediated tomato (Solanum lycopersicum) fruit coloring.Communications biology · 2026Article
- Signaling mechanisms governing the environmental modulation of fruit quality development.Horticulture research · 2026Article
- Melatonin as a Pre- and Postharvest Tool for Enhancing Fruit Quality.Plants (Basel, Switzerland) · 2026Review
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Authors and funding
8 authors.
Funding
Abstract
Fleshy Fruit (FF) ripening is regulated by multiple hormones, which can be categorized into two groups, i.e., the positive signals acting to promote FF ripening and the negative signals acting to suppress FF ripening. Ethylene (ET) and abscisic acid (ABA) are two predominant positive signals respectively controlling climacteric (CL) and non-climacteric (NC) FF ripening, whereas auxin (IAA) is the predominant negative signal controlling both FF growth and ripening. Functioning of these hormones is initiated by an alteration of the hormonal levels, which is referred to as the process of Hormonal Signal Production (HSP) in FF development and ripening. While the hormonal regulation of FF ripening has been extensively studied and reviewed, knowledge of HSP has never been summarized and discussed. The purpose of this review is to summarize and discuss the triggering mechanism of HSP. We first summarize the physiological, biochemical and molecular bases of HSP for three crucial hormones, ET, ABA, and IAA, including hormonal metabolism, transport and reciprocal regulation of HSP among different hormones, we then summarize and discuss the recent discoveries on the mechanism of cellular signal transduction of HSP. Finally, we propose several viewpoints to facilitate comprehension of the future research endeavors.
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Registered trials
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