ArticleCureus2025
Success of Coenzyme Q10 in Treating Steroid-Resistant Nephrotic Syndrome in Jordan: A Case Series.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Uncommon Factors Leading to Nephrotic Syndrome.Biomedicines · 2025Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
9 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Focal segmental glomerulosclerosis (FSGS) is one of the leading causes of primary end-stage kidney disease in the pediatric age group. It is commonly associated with steroid-resistant nephrotic syndrome (SRNS), which ultimately leads to impairment in the function of the glomerular filtration system. Genetic studies have revealed nearly 50 types of gene deficiency disorders linked to the development of both FSGS and SRNS. Among these disorders, primary coenzyme Q10 (CoQ10) deficiency is classified as one of the few types that respond well to treatment. CoQ10 plays a crucial role within the mitochondria, including energy production through the electron transport chain. A network of at least 17 genes is necessary for its synthesis. When mutations occur in the genes responsible for CoQ10 production, a deficiency can develop, leading to mitochondrial dysfunction and reduced cellular energy levels. Since CoQ10 is crucial for mitochondrial function, its deficiency has been recognized as a potential therapeutic target. Increasing evidence suggests that CoQ10 supplementation may provide clinical benefit in treating this condition. We present three pediatric cases of SRNS that did not respond to standard treatment. Despite kidney biopsies revealing FSGS, conventional therapies proved ineffective. The patients were started on CoQ10 supplementation, which led to a complete resolution of nephrotic syndrome. Their kidney function remained within the normal range during follow-up, and proteinuria remained stable, indicating a sustained therapeutic response.
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Registered trials
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