Article in MedComm, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
10 authors.
Hengkai HeGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Mingjing HaoGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Piao LuoGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Junhui ChenDepartment of Pulmonary and Critical Care Medicine Shenzhen Institute of Respiratory Diseases and Shenzhen Clinical Research Centre for Geriatrics Shenzhen People's Hospital First Affiliated Hospital of Southern University of Science and Technology Second Clinical Medical College of Jinan University Shenzhen Guangdong China.
Yehai AnGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Jingnan HuangDepartment of Pulmonary and Critical Care Medicine Shenzhen Institute of Respiratory Diseases and Shenzhen Clinical Research Centre for Geriatrics Shenzhen People's Hospital First Affiliated Hospital of Southern University of Science and Technology Second Clinical Medical College of Jinan University Shenzhen Guangdong China.
Ruiyi HeGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Qingfeng DuGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Qian ZhangGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.
Jigang WangGuangdong Provincial Key Laboratory of Chinese Medicine Pharmaceutics School of Traditional Chinese Medicine and School of pharmaceutical Sciences Southern Medical University Guangzhou China.ORCID https://orcid.org/0000-0002-0575-0105
Funding
No grant is acknowledged in the PubMed record.
6 · The paper itself
Abstract
Rheumatoid arthritis (RA), a prevalent and incurable autoimmune disease globally, is characterized by the immune system attacking the body's own tissues, leading to joint inflammation and damage. Capsaicin (CAP), from
Indexed as
activity‐based protein profilingantioxidantcapsaicinperoxiredoxin‐2rheumatoid arthritis
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Inhibition Peroxiredoxin-2 by Capsaicin Ameliorates Rheumatoid Arthritis via ROS-Mediated Apoptosis in Fibroblast-Like Synoviocytes. · full record | OpenQuestion