ArticleCureus2025
Fulminant Influenza Myocarditis Requiring Extracorporeal Membrane Oxygenation (ECMO) Support.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Viral infections may lead to myocarditis, which is inflammation of the myocardium. This inflammation, when severe enough, can result in left ventricular dysfunction and potentially reduce the left ventricular ejection fraction (LVEF). In rare cases, the effects of this inflammation lead to hemodynamic changes that can be life-threatening. We discuss a case of a 38-year-old female recently diagnosed with influenza A (H3 subtype) who presented to our institution's emergency department for evaluation after an episode of syncope, as well as intermittent chest pressure and dyspnea on exertion. Initial vitals displayed a heart rate of 87 bpm and blood pressure of 105/66 mmHg. The physical examination demonstrated a regular rhythm, no lower extremity edema, and lungs that were clear to auscultation. She was found to have an elevated pro-B-type natriuretic peptide level of 6152 pg/mL and a positive influenza A polymerase chain reaction (PCR) test. A transthoracic echocardiogram (TTE) was obtained and demonstrated globally reduced left ventricular systolic function with an estimated ejection fraction of 28%, as well as reduced right ventricular systolic function. Over the next six hours, the patient became progressively tachycardic and hypotensive, with a heart rate of 135 bpm and a blood pressure measured at 46/28 mmHg. She was initially admitted to the cardiovascular ICU and started on dobutamine and vasopressin. Pulmonary artery catheterization was completed for better evaluation of cardiogenic shock, and it demonstrated a severely reduced cardiac index of 0.9 L/min/m
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