Evidence map›Paper›PMID 40428340›Full record

ArticleGenes2025

The rs1403543 Polymorphism of

Iwona Gorący, Karol Miler, Klaudyna Lewandowska, Monika Rychel, Beata Łoniewska, Andrzej Ciechanowicz

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Genes, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Iwona GorącyDepartment of Clinical and Molecular Biochemistry, Pomeranian Medical University, 70-111 Szczecin, Poland.
Karol MilerDepartment of Clinical and Molecular Biochemistry, Pomeranian Medical University, 70-111 Szczecin, Poland.
Klaudyna LewandowskaDepartment of Clinical and Molecular Biochemistry, Pomeranian Medical University, 70-111 Szczecin, Poland.ORCID 0000-0002-6732-2341
Monika RychelDepartment of Clinical and Molecular Biochemistry, Pomeranian Medical University, 70-111 Szczecin, Poland.
Beata ŁoniewskaDepartment of Neonatal Diseases, Pomeranian Medical University, 70-111 Szczecin, Poland.
Andrzej CiechanowiczDepartment of Clinical and Molecular Biochemistry, Pomeranian Medical University, 70-111 Szczecin, Poland.ORCID 0000-0002-3804-440X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BACKGROUND/

objectivesLeft ventricular hypertrophy is a significant independent risk factor for increased cardiovascular morbidity and mortality. There are some reports indicating an association of rs1403543 (1675G>A) polymorphism in the

methodsThe study group comprised 207 consecutive, full-term, healthy newborns. LVM was assessed, on the 3rd day after birth, from the M-mode echocardiographic measurements of left ventricular dimensions using the Penn convention, with the Huwez et al.-modified equation mode. The

resultsThere were no significant differences in clinical and echocardiographic characteristics of male newborns in regard to the

conclusionsOur results suggest that the

Indexed as

Heart VentriclesHypertrophy, Left VentricularPolymorphism, Single NucleotideReceptor, Angiotensin, Type 2AllelesEchocardiographyFemaleHumansInfant, NewbornMalePolandReceptor, Angiotensin, Type 2genetic polymorphismleft ventricular massnewbornstype 2 receptor for angiotensin II

Identifiers

PMID40428340
PMCPMC12111044

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.