ReviewCureus2025
Lateral Spread Response in Hemifacial Spasm: Physiological Mechanisms, Intraoperative Utility, and Prognostic Significance.
Review in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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0 citing papers in PubMed.
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Facial nerve compression by blood vessels near the brainstem can cause hemifacial spasm (HFS). There are two treatment options for this condition: botulinum toxin and surgical microvascular decompression (MVD). During microvascular decompression, the facial nerve is separated from the offending vessel, and intraoperative neuromonitoring in these patients demonstrates abnormal muscle response (AMR), which is known as the lateral spread response (LSR). Though the disappearance of lateral spread response is a hallmark of successful microvascular decompression, little information is available about its physiological origin and diagnostic utility. In the present review, we have attempted to address the aforementioned caveats about lateral spread response with an emphasis on the intraoperative utility and diagnostic role of this electrophysiological phenomenon.
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