ArticleOrthopaedic journal of sports medicine2025
Patellofemoral Cartilage Degradation Based on T1ρ Relaxation Times Varies Inversely With BMI After Patellar Dislocation.
Article in Orthopaedic journal of sports medicine, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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Who cites it
1 citing paper in PubMed.
- Associations Between Patient Characteristics and Cartilage T1ρ Relaxation Times Vary Over Time Following Patellar Dislocation.Journal of orthopaedic research : official publication of the Orthopaedic Research Society · 2026Article
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8 authors.
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Abstract
Background: Elevated body mass index (BMI) is a primary factor contributing to osteoarthritis of the knee, but data are lacking on the relationship between BMI and cartilage properties shortly after a traumatic injury. Purpose/Hypothesis: The purpose of the study was to relate BMI to patellofemoral cartilage properties after a patellar dislocation, using magnetic resonance imaging-based T1ρ relaxation times to assess cartilage. It was hypothesized that high BMI is correlated with long patellofemoral cartilage T1ρ relaxation times, indicating cartilage degradation, in both control knees and knees after patellar dislocation. Study Design: Cross-sectional study; Level of evidence, 3. Methods: T1ρ relaxation time mapping was performed for underweight to obese (BMI < 40 kg/m Results: For the control group, long T1ρ relaxation times were significantly correlated with high BMI for every region of patellofemoral cartilage except the lateral patella ( Conclusion: Long patellofemoral cartilage T1ρ relaxation times were related to high BMI for healthy knees, but unexpectedly related to low BMI after patellar dislocation. The results do not indicate a heightened risk of progressive cartilage degradation to osteoarthritis for overweight and obese patients immediately after patellar dislocation. For patients at a healthy weight and underweight, the traumatic injury is associated with early cartilage degradation, which could potentially initiate progressive degradation to posttraumatic osteoarthritis.
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