Evidence map›Paper›PMID 40399475›Full record

ArticleMammalian genome : official journal of the International Mammalian Genome Society2025

Snrnp25 is a candidate for the peri-implantation lethal phenotype of the Hba deletions.

Ana María Velásquez-Escobar, Andrew E Hillhouse, Terry Magnuson, David W Threadgill

Abstract read
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In one paragraph

Article in Mammalian genome : official journal of the International Mammalian Genome Society, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Ana María Velásquez-EscobarDepartment of Cell Biology and Genetics, Texas A&M University, College Station, TX, 77843, USA.
Andrew E HillhouseDepartment of Cell Biology and Genetics, Texas A&M University, College Station, TX, 77843, USA.
Terry MagnusonDepartment of Genetics, University of North Carolina, Chapel Hill, NC, 27599, USA.
David W ThreadgillDepartment of Cell Biology and Genetics, Texas A&M University, College Station, TX, 77843, USA. dwt@tamu.edu.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mutations in adult hemoglobin alpha genes in humans lead to blood disorders commonly known as α-thalassemia. In search of a mouse model for this disease, mutagenesis screens have identified several deletions that resemble these phenotypes. The Hba

Indexed as

alpha-ThalassemiaGene Deletionalpha-GlobinsAnimalsErbB ReceptorsFemaleHomozygoteHumansMicePhenotypealpha-GlobinsErbB ReceptorsDeletion complexEmbryonic lethalMouse modelThalassemia

Identifiers

PMID40399475
PMCPMC12408735

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.