In one paragraphArticle in Molecular and cellular biochemistry, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
9 authors.
Izabella LiceGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.ORCID http://orcid.org/0000-0003-2310-2010 Henrique T K ItoGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.
Rebeca D Correia-SilvaGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.ORCID http://orcid.org/0000-0002-9533-9781 Diego D SantosGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.ORCID http://orcid.org/0000-0002-1796-9577 Gisela R S SassoGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.ORCID http://orcid.org/0000-0002-6583-1329 Paulo C FrancoGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.
Karin V GrecoGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil.ORCID http://orcid.org/0000-0002-6261-7650 Cristiane D GilGraduate Program in Structural and Functional Biology, Department of Morphology and Genetics, Paulista School of Medicine (EPM), Federal University of São Paulo (UNIFESP), São Paulo, SP, Brazil. cristiane.gil@unifesp.br.ORCID http://orcid.org/0000-0001-6979-4126 Funding
Conselho Nacional de Desenvolvimento Científico e Tecnológico 308524/2022-5Coordenação de Aperfeiçoamento de Pessoal de Nível Superior 88881.310737/2018-01Fundação de Amparo à Pesquisa do Estado de São Paulo 20/03565-2Fundação de Amparo à Pesquisa do Estado de São Paulo 21/01541-1Fundação de Amparo à Pesquisa do Estado de São Paulo 21/06059-3
6 · The paper itselfAbstract
Inflammatory bowel disease (IBD) remains a complex and multifaceted condition, with its management dependent on a thorough understanding of its underlying mechanisms. While the ANXA1-FPR axis is implicated in the pathogenesis of IBD, its relationship with the NLRP3 inflammasome in this context has not been established. Thus, this study aimed to elucidate the intricate relationship between ANXA1, FPRs, and the NLRP3 inflammasome in the pathogenesis of IBD. For this purpose, mRNA and protein expression of ANXA1, FPRs, and NLRP3 inflammasome were analyzed using transcriptomic data (GSE179285) and immunohistochemistry on ileum and colon samples from CD patients and healthy controls. In vitro, ANXA1-derived peptide Ac
Indexed as
Annexin A1Crohn DiseaseInflammasomesNLR Family, Pyrin Domain-Containing 3 ProteinPeptidesAdultCaco-2 CellsFemaleHumansMaleReceptors, Formyl PeptideReceptors, LipoxinAnnexin A1annexin A1 peptide (2-26)ANXA1 protein, humanFPR2 protein, humanInflammasomesNLR Family, Pyrin Domain-Containing 3 ProteinNLRP3 protein, humanPeptidesReceptors, Formyl PeptideReceptors, LipoxinAc2–26ANXA1FPRInflammatory bowel diseaseNLRP3 inflammasome
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