Evidence map›Paper›PMID 40396394›Full record

ArticleTraffic (Copenhagen, Denmark)2025

A Sensitive and Versatile Cell-Based Assay Combines Luminescence and Trapping Approaches to Monitor Unconventional Protein Secretion.

Morgane Denus, Aurore Filaquier, William Fargues, Eloïse Néel, Sarah E Stewart, Maëlle Colladant, Thomas Curel, Alexandre Mezghrani, Philippe Marin, Sylvie Claeysen and 3 more

Abstract read
In one paragraph

Article in Traffic (Copenhagen, Denmark), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Review
  2. Review
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  6. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Morgane DenusInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
Aurore FilaquierInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
William FarguesInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.ORCID 0009-0005-5291-0828
Eloïse NéelInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
Sarah E StewartDepartment of Biochemistry and Chemistry, School of Agriculture, Biomedicine and Environment, La Trobe Institute for Molecular Science, La Trobe University, Melbourne, Victoria, Australia.
Maëlle ColladantInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
Thomas CurelInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
Alexandre MezghraniCentre de Biologie Structurale (CBS), University of Montpellier, CNRS, INSERM, Montpellier, France.ORCID 0000-0003-3719-9933
Philippe MarinInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.ORCID 0000-0002-5977-7274
Sylvie ClaeysenInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
David C RubinszteinDepartment of Medical Genetics, University of Cambridge, Cambridge Institute for Medical Research, Cambridge, UK.
Marie-Laure ParmentierInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.
Julien VilleneuveInstitute of Functional Genomics (IGF), University of Montpellier, CNRS, INSERM, Montpellier, France.ORCID 0000-0002-5430-1680

Funding

Agence Nationale de la Recherche ANR-23-CE16-0012Agence Nationale de la Recherche ANR-24-CE16-5161-02Association France ParkinsonAustralian Research CouncilCentre National de la Recherche Scientifique (CNRS)Centre of Excellence in Neurodegeneration (CoEN)Fédération pour la Recherche sur le CerveauFondation pour la Recherche Médicale (FRM) MND202310017892Fondation Vaincre AlzheimerFrance Alzheimer et maladies apparentéesFrench National Research Agency ANR-10_INBS-04Institut National pour la Santé et la Recherche Médicale (INSERM)National Institute for Health Research Cambridge Biomedical Research CentreUK Dementia Research InstituteUK Medical Research CouncilUniversité de Monpellier
6 · The paper itself

Abstract

In addition to the conventional endoplasmic reticulum (ER)-Golgi secretory pathway, alternative routes are increasingly recognized for their critical roles in exporting a growing number of secreted factors. These alternative processes, collectively referred to as unconventional protein secretion (UcPS), challenge traditional views of protein and membrane trafficking. Unlike the well-characterized molecular machinery of the conventional secretory pathway, the mechanisms underlying UcPS remain poorly understood. Various UcPS pathways may involve direct transport of cytosolic proteins across the plasma membrane or the incorporation of cargo proteins into intracellular compartments redirected for secretion. Identifying the specific chaperones, transporters and fusion machinery involved in UcPS cargo recognition, selection and transport is crucial to decipher how cargo proteins are selectively or synergistically directed through multiple secretory routes. These processes can vary depending on cell type and in response to particular stress conditions or cellular demands, underscoring the need for standardized tools and methods to study UcPS. Here, we combine the sensitivity of split NanoLuc Binary Technology with the versatility of the Retention Using Selective Hooks (RUSH) system to develop a straightforward and reliable cell-based assay for investigating both conventional and unconventional protein secretion. This system allows for the identification of intracellular compartments involved in UcPS cargo trafficking. Additionally, its sensitivity enabled us to demonstrate that disease-associated mutants or variants of Tau and superoxide dismutase-1 (SOD1) show altered secretion via UcPS. Finally, we leveraged this assay to screen for Alzheimer's disease risk factors, revealing a functional link between amyloid-beta production and Tau UcPS. This robust assay provides a powerful tool for increasing our knowledge of protein secretion mechanisms in physiological and pathological contexts.

Indexed as

Protein TransportEndoplasmic ReticulumHumansSecretory PathwaySuperoxide Dismutase-1tau ProteinsSOD1 protein, humanSuperoxide Dismutase-1tau Proteinsintercellular communicationintracellular compartmentsneurodegenerative diseasesprotein traffickingsecretory pathwaysplit luciferasetauunconventional protein secretion

Identifiers

PMID40396394
PMCPMC12093449

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.