ArticleVeterinary research communications2025
Zoonotic relevance of multidrug-resistant bacteria in parrots with respiratory illness.
Article in Veterinary research communications, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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Who cites it
5 citing papers in PubMed.
- Draft genomes of two multidrug-resistantMicrobiology resource announcements · 2026Article
- Detection and Characterization of ESBL-Producing and Carbapenem-Resistant Klebsiella pneumoniae in Ornamental Birds and Their Surrounding Environments.MicrobiologyOpen · 2026Article
- Virulence determinants and multidrug resistance of emerging Salmonella enterica subspecies salamae in broiler chickens: a potential zoonotic concern.Veterinary research communications · 2026Article
- Review
- Veterinarians' Perspectives on the Antimicrobial Resistance (AMR) Dashboard: A Survey of Needs and Preferences to Inform Development.Veterinary sciences · 2025Article
Corrections and comments
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Authors and funding
9 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Nowadays, research attention is paid to the investigation of bacterial pathogens in the cloaca of parrots rather than the nasal niche, which is largely ignored. Therefore, this study aimed to investigate the nasal carriage of multidrug-resistant bacteria with zoonotic potential in parrots suffering from respiratory illness. Nasal swabs were collected from 75 sick parrots with respiratory illness, and they were subjected to microbiological isolation and identification, followed by antimicrobial susceptibility testing. Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, and Staphylococcus aureus were isolated with a prevalence rate of 36%, 32%, 26.7%, and 9.3%, respectively, while one isolate (1.3%) of Staphylococcus pseudointermedius, Staphylococcus simulans, Staphylococcus sciuri, and Enterococcus faecalis was identified. E. coli, K. pneumoniae, and P. mirabilis were investigated for ESBL genes, Staphylococcus species for the mecA gene, followed by SCCmec typing, and E. faecalis for the vanA and vanB genes. Regarding beta-lactamase-encoding genes, bla
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