In one paragraphArticle in Environmental science & technology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
12 authors.
Alexander R ColeDepartment of Environmental Science, Center for Reservoir and Aquatic Systems Research, Baylor University, Waco, Texas 76706, United States.ORCID 0000-0003-4608-2518 Gerald T AnkleyGreat Lakes Toxicology and Ecology Division, US Environmental Protection Agency, Duluth, Minnesota 55804, United States.ORCID 0000-0002-9937-615X Jenna E CavallinGreat Lakes Toxicology and Ecology Division, US Environmental Protection Agency, Duluth, Minnesota 55804, United States.ORCID 0000-0001-7883-4740 Jacob R CollinsGreat Lakes Toxicology and Ecology Division, Oak Ridge Institute for Science and Education, US EPA, Duluth, Minnesota 55804, United States.
Kathleen M JensenGreat Lakes Toxicology and Ecology Division, US Environmental Protection Agency, Duluth, Minnesota 55804, United States.
Michael D KahlGreat Lakes Toxicology and Ecology Division, US Environmental Protection Agency, Duluth, Minnesota 55804, United States.
Alex J KasparekGreat Lakes Toxicology and Ecology Division, Oak Ridge Institute for Science and Education, US EPA, Duluth, Minnesota 55804, United States.
Ba Reum KwonDepartment of Environmental Science, Center for Reservoir and Aquatic Systems Research, Baylor University, Waco, Texas 76706, United States.
Yesmeena M ShmaitellyDepartment of Environmental Science, Center for Reservoir and Aquatic Systems Research, Baylor University, Waco, Texas 76706, United States.
Laura M LanganDepartment of Environmental Science, Center for Reservoir and Aquatic Systems Research, Baylor University, Waco, Texas 76706, United States.ORCID 0000-0002-2095-7163 Daniel L VilleneuveGreat Lakes Toxicology and Ecology Division, US Environmental Protection Agency, Duluth, Minnesota 55804, United States.ORCID 0000-0003-2801-0203 Bryan W BrooksDepartment of Environmental Science, Center for Reservoir and Aquatic Systems Research, Baylor University, Waco, Texas 76706, United States.ORCID 0000-0002-6277-9852 Funding
The effects of climate change on the natural attenuation and sedimentary record of harmful algal bloom toxins and microbial signalsP01ES028942 · NIEHS · UNIVERSITY OF SOUTH CAROLINA AT COLUMBIA · PI CHATTERJEE, SAURABH · 2018 to 2022
$5.7MIntramural EPA EPA999999NIEHS NIH HHS P01 ES028942
6 · The paper itselfAbstract
Despite structural and functional conservation across vertebrate species, the glucocorticoid receptor has been minimally studied in comparison to other biological targets for endocrine-disrupting compounds in aquatic systems. Because prolonged use of pharmaceutical glucocorticoids in humans has been linked to osteoporosis and impaired bone growth, we hypothesized that the ability of teleost fish to regenerate fins following damage may be inhibited by exposure to synthetic glucocorticoids in the environment. In the present study, we examined fin regeneration following a 7 days waterborne exposure of juvenile fathead minnows (
Indexed as
Animal FinsCyprinidaeGlucocorticoidsRegenerationAnimalsDexamethasoneFluticasoneDexamethasoneFluticasoneGlucocorticoidsadverse outcome pathwaydexamethasonefin regenerationfishfluticasone propionate
Identifiers
PMID40326831
PMCPMC13086407
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