ArticleWorld journal of cardiology2025
High-output heart failure secondary to iatrogenic arteriovenous fistula: A case report.
Article in World journal of cardiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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Who cites it
4 citing papers in PubMed.
- Misdiagnosis of restrictive cardiomyopathy averted by a stethoscope: a case report.European heart journal. Case reports · 2026Article
- Article
- Resolution of a femoral arteriovenous fistula secondary to chronic Iliac vein occlusion after stenting: a case report.BMC surgery · 2026Article
- High-Output Heart Failure from A Decade-Old Traumatic AV Fistula: A Case Report.International medical case reports journal · 2026Article
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3 authors.
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Abstract
backgroundArteriovenous fistula is a rare cause of refractory heart failure, and corrective measures may lead to dramatic improvement; however, the long-term cardiac remodeling outcomes, particularly after delayed closure, remain unclear. CASE SUMMARY: A 57-year-old man was admitted to the hospital with complaints of exertional dyspnea for more than 10 years. Physical examination revealed wet crackles in the lungs and a continuous machinery murmur in the left lower back and groin area. Asymmetric edema and varicose veins were observed in the lower limbs. Echocardiography revealed a dilated right ventricle with severe pulmonary hypertension. Computed tomography revealed a left common iliac arteriovenous fistula linked to prior lumbar disc surgery. Surgical repair resolved the symptoms, with echocardiography at 4 months showing a reduced right atrium (RA) and ventricular (RV) diameter and tricuspid regurgitation. However, during the 2-year follow-up, gradual RA and RV re-expansion (from 35 mm to 51 mm and from 26 mm to 46 mm, respectively) was observed, despite sustained clinical stability.
conclusionThis case highlights that delayed arteriovenous fistula closure may result in incomplete right heart reverse remodeling, even after symptomatic relief. Potential mechanisms include persistent hemodynamic stress from subclinical residual shunting or functional impairment due to chronic volume overload. Early intervention before irreversible right heart damage is critical for optimal outcomes.
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