ReviewFrontiers in neurology2025
Research progress in Pusher Syndrome after stroke.
Review in Frontiers in neurology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
4 citing papers in PubMed.
- The clinical effects and cortical mechanism of rTMS in poststroke lateropulsion: a randomized controlled trial.BMC medicine · 2026Trial
- Progress in the application of virtual reality technology in the rehabilitation of patients with pusher syndrome after stroke.Frontiers in neurologyReview
- Effects and central mechanisms of acupuncture for post-stroke vascular vertigo: study protocol of a multicenter, randomized, sham-controlled trial.Frontiers in neurologyArticle
- Advances in music therapy for neurological rehabilitation.Frontiers in neurologyReview
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Post-stroke Pusher Syndrome is a postural control disorder. It is characterized by active tilting toward the hemiplegic side and resistance to correction. This significantly impacts patients' motor function and quality of life. Its incidence varies greatly due to different research designs and assessment criteria. Literature reports an incidence ranging from 5% to 63%, and the incidence in patients with right brain damage (17.4%) is much higher than that in patients with left brain damage (9.5%). Etiological studies indicate that damage to the parietal lobe, thalamus, insula, and postcentral gyrus is the main pathological basis. The key mechanism is the interruption of thalamocortical connections. Typical clinical manifestations include trunk tilting in supine position, asymmetric weight-bearing in sitting, weight shift in standing, and impaired weight transfer during gait. Patients often have unilateral spatial neglect, which exacerbates balance disorders. Prognosis shows about 90% of patients recover within 6 months, but 10% to 15% may have long-term symptoms. Early rehabilitation intervention can significantly improve functional outcomes. This article comprehensively reviews the nomenclature, incidence, etiology, lesion sites, clinical manifestations, and prognosis of Pusher Syndrome, providing a research foundation for future studies on post-stroke Pusher Syndrome.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.