Evidence map›Paper›PMID 40302691›Full record

ArticleHistology and histopathology2025

Period 1 (

Shuko Hata, Hiroki Shimada, Atsushi Yokoyama, Yasuhiro Nakamura

Abstract read
PubMed Publisher
In one paragraph

Article in Histology and histopathology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Shuko HataDivision of Pathology, Faculty of Medicine, Tohoku Medical and Pharmaceutical University, Fukumuro, Miyagino-ku, Sendai, Miyagi, Japan.
Hiroki ShimadaDivision of Pathology, Faculty of Medicine, Tohoku Medical and Pharmaceutical University, Fukumuro, Miyagino-ku, Sendai, Miyagi, Japan.
Atsushi YokoyamaDepartment of Molecular Endocrinology, Tohoku University Graduate School of Medicine, Seiryo-machi, Aoba-ku, Sendai, Miyagi, Japan.
Yasuhiro NakamuraDivision of Pathology, Faculty of Medicine, Tohoku Medical and Pharmaceutical University, Fukumuro, Miyagino-ku, Sendai, Miyagi, Japan.

Funding

JSPS KAKENHI JP18K06995
6 · The paper itself

Abstract

Glucocorticoid receptor (GR) has been implicated in prostate carcinoma and possibly involved in cancer growth and progression. Therefore, in this study, we examined whether the genes involved in cell proliferation regulation were induced by cortisol in androgen-independent human prostate cancer DU145 cells. Gene expression profiling of the DU145 cell pathway was conducted using the RT

Indexed as

Cell ProliferationHydrocortisonePeriod Circadian ProteinsProstatic NeoplasmsAndrogensCell Line, TumorDexamethasoneGene Expression ProfilingGene Expression Regulation, NeoplasticGlucocorticoidsHumansMaleReceptors, GlucocorticoidAndrogensDexamethasoneGlucocorticoidsHydrocortisonePER1 protein, humanPeriod Circadian ProteinsReceptors, Glucocorticoid

Identifiers

PMID40302691

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.