Evidence map›Paper›PMID 40285622›Full record

ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2025

Mas Signaling Potentiates Neutrophil Extracellular Traps Formation Induced by Endothelial Cells Derived S1P in Mice with Acute Liver Failure.

Bo Yang, Shuai Chen, Xiaoqi Xia, Ziwen Tao, Chun Liu, Shanshan Li, Shuo Zhang, Jiali Huang, Lu Xia, Wenqiang Quan and 2 more

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Article
  2. Article
  3. CD177Gut · 2026
    Article
  4. Article
  5. The gut as a central hub for multi-organ crosstalk in aging.Cellular and molecular life sciences : CMLS · 2026
    Review
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Bo YangDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Shuai ChenDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Xiaoqi XiaDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Ziwen TaoDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Chun LiuDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Shanshan LiDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Shuo ZhangDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Jiali HuangDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Lu XiaDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Wenqiang QuanDepartment of Laboratory Medicine, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.
Changqing YangDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.ORCID https://orcid.org/0000-0001-6997-0214
Jing LiDepartment of Gastroenterology and Hepatology, Tongji Hospital, School of Medicine, Tongji University, Shanghai, 200065, China.ORCID https://orcid.org/0000-0002-6815-495X

Funding

China Postdoctoral Science Foundation 2023M732649China Postdoctoral Science Foundation 2023M732653National Natural Science Foundation of China 82300702National Natural Science Foundation of China 82370627National Natural Science Foundation of China U22A20275Shanghai Municipal Science and Technology Commission 22YF1442200Shanghai Municipal Science and Technology Commission 23ZR1457900Shanghai Pujiang Program 23PJD083
6 · The paper itself

Abstract

Mas, a newly identified G-protein-coupled receptor, is prevalent in myeloid-derived immune cells and plays a key role in inflammation. This study investigates Mas signaling and neutrophil extracellular traps (NETs) in acute liver failure (ALF), aiming to elucidate their mechanisms. Male Mas1

Indexed as

Endothelial CellsExtracellular TrapsLiver Failure, AcuteLysophospholipidsNeutrophilsReceptors, G-Protein-CoupledSphingosineAnimalsDisease Models, AnimalMaleMiceMice, Inbred C57BLMice, KnockoutSignal TransductionLysophospholipidsReceptors, G-Protein-CoupledSphingosinesphingosine 1-phosphateendothelial cellsfarnesoid X receptorMasneutrophil extracellular trapssphingosine 1‐phosphate

Identifiers

PMID40285622
PMCPMC12120760

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.