Evidence map›Paper›PMID 40258227›Full record

ArticlePLoS computational biology2025

The NOSTRA model: Coherent estimation of infection sources in the case of possible nosocomial transmission.

David J Pascall, Christopher Jackson, Stephanie Evans, Theodore Gouliouris, Christopher J R Illingworth, Stefan G Piatek, Julie V Robotham, Oliver Stirrup, Ben Warne, Judith Breuer and 1 more

Abstract read
In one paragraph

Article in PLoS computational biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

David J PascallMRC Biostatistics Unit, University of Cambridge, Cambridge, United Kingdom.ORCID 0000-0002-7543-0860
Christopher JacksonMRC Biostatistics Unit, University of Cambridge, Cambridge, United Kingdom.
Stephanie EvansHCAI, Fungal, AMR, AMU & Sepsis Division, UK Health Security Agency, London, United Kingdom.
Theodore GouliourisClinical Microbiology and Public Health Laboratory, Cambridge University Hospitals NHS Foundation Trust, Cambridge, United Kingdom.
Christopher J R IllingworthMRC-University of Glasgow Centre for Virus Research, University of Glasgow, Glasgow, United Kingdom.
Stefan G PiatekAdvanced Research Computing, University College London, London, United Kingdom.ORCID 0000-0002-3028-5292
Julie V RobothamHCAI, Fungal, AMR, AMU & Sepsis Division, UK Health Security Agency, London, United Kingdom.
Oliver StirrupInstitute for Global Health, University College London, London, United Kingdom.ORCID 0000-0002-8705-3281
Ben WarneClinical Microbiology and Public Health Laboratory, Cambridge University Hospitals NHS Foundation Trust, Cambridge, United Kingdom.
Judith BreuerInfection, Immunity and Inflammation Department, Great Ormond Street Institute of Child Health, University College London, London, United Kingdom.
Daniela De AngelisMRC Biostatistics Unit, University of Cambridge, Cambridge, United Kingdom.

Funding

Department of Health and Social CareMedical Research Council MC\UU\00002/11Medical Research Council MC\UU\00034/1NIHR NIHR200652
6 · The paper itself

Abstract

Nosocomial, or hospital-acquired, infections are a key determinant of patient health in healthcare facilities, leading to longer stays and increased mortality. In addition to the direct effects on infected patients, the burden imposed by nosocomial infections impacts both staff and other patients by increasing the load on the healthcare system. The appropriate infection control response may differ depending on whether the infection was acquired in the hospital or the community. For example, nosocomial outbreaks may require ward closures to reduce the risk of onward transmission, whilst this may not be an appropriate response to repeated importations of infections from outside the facility. Unfortunately, it is often unclear whether an infection detected in a healthcare facility is nosocomial, as the time of infection is unobserved. Given this, there is a strong case for the development of models that can integrate multiple datasets available in hospitals to assess whether an infection detected in a hospital is nosocomial. When assessing nosocomiality, it is beneficial to take into account both whether the timing of infection is consistent with hospital acquisition and whether there are any likely candidates within the hospital who could have been the source of the infection. In this work, we developed a Bayesian model which jointly estimates whether a given infection detected in hospital is nosocomial and whether it came from a set of individuals identified as candidates by hospital staff. The model coherently integrates pathogen genetic information, the timings of epidemiological events, such as symptom onset, and location data on the infected patient and candidate infectors. We illustrated this model on a real hospital dataset showing both its output and how the impact of the different data sources on the assessed probabilities are contingent on what other data has been included in the model, and validated the calibration of the predictions against simulated data.

Indexed as

Cross InfectionModels, BiologicalBayes TheoremComputational BiologyDisease OutbreaksHumans

Identifiers

PMID40258227
PMCPMC12121921

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.