Evidence map›Paper›PMID 40243809›Full record

ArticleInternational journal of molecular sciences2025

Characterization of Protein Expression and Signaling Pathway Activation That May Contribute to Differential Biological Functions in Porcine Arterial and Venous Smooth Muscle Cells.

Eyla C Arteaga, Christine Wai, Unimunkh Uriyanghai, Medha Rudraraju, Huanjuan Su, Vinay A Sudarsanam, Samuel O'Brien Haddad, John S Poulton, Prabir Roy-Chaudhury, Gang Xi

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Eyla C ArteagaUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
Christine WaiUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
Unimunkh UriyanghaiUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
Medha RudrarajuDepartment of Biology, Drexel University, Philadelphia, PA 19104, USA.
Huanjuan SuUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.ORCID 0009-0000-2822-5875
Vinay A SudarsanamUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
Samuel O'Brien HaddadUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
John S PoultonUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
Prabir Roy-ChaudhuryUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.
Gang XiUNC Kidney Center, Division of Nephrology and Hypertension, Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA.

Funding

Pathobiology of ANCA Glomerulonephritis: Targeting Adaptive and Innate Immune Processes for Precision TherapiesR01DK125350 · NIDDK · UNIV OF NORTH CAROLINA CHAPEL HILL · PI CIAVATTA, DOMINIC J, FALK, RONALD J · 2020 to 2024
$3.4M
Modulation of VSMC phenotype through the Insulin Receptor Substrate-1/Kruppel-like factor-4 signal transduction pathway: a Novel Target for AVF DysfunctionR01DK132328 · NIDDK · UNIV OF NORTH CAROLINA CHAPEL HILL · PI PRABIR ROY-CHAUDHURY, Gang Xi · 2022 to 2026
$2.7M
NIDDK NIH HHS R01 DK125350NIDDK NIH HHS R01 DK132328NIH HHS 1R01DK132328-03A5
6 · The paper itself

Abstract

Arteriovenous fistulae (AVF) are the preferred mode of dialysis vascular access but have a maturation failure rate of over 50%. Venous segment stenosis is one of the main reasons. However, the reasons for the prevalence of stenosis in the venous segment, as opposed to the arterial segment, remain unknown. We hypothesize that differences in the protein expression profiles and in the activation of key signaling pathways of the vascular smooth muscle cells (VSMCs) in these two segments may contribute to this difference. In this study, arterial porcine VSMCs (ApSMCs) and venous porcine VSMCs (VpSMCs) were isolated to examine relevant protein expression and cell signaling pathway activation via Western blots and immunofluorescent staining. Our results revealed that growth-medium-stimulated cell proliferation was significantly higher in VpSMCs compared to ApSMCs, but no difference was detected in PDGF-BB-stimulated proliferation. VpSMCs migration was significantly greater than ApSMCs under both the serum-free condition and in the growth medium condition. In addition, VpSMCs appeared more susceptible to PDGF-BB-stimulated cell dedifferentiation. Furthermore, P53, vitronectin, collagen 1A1 and integrin β3 had lower expression in VpSMCs, whereas fibronectin, FAK, VCAM-1, MMP-9, TIMP-2 and TIMP-3 had higher expression as compared to ApSMCs. Meanwhile, PDGF-BB stimulated P38, JNKs and AKT activation, which were more potent in VpSMCs. Our results identify significant differences between VpSMCs and ApSMCs in the expression of functional proteins and the activation of signaling pathways, which may be responsible for more aggressive venous-as compared to arterial-stenosis in the clinical setting of arteriovenous access or other similar procedures.

Indexed as

ArteriesMuscle, Smooth, VascularMyocytes, Smooth MuscleSignal TransductionVeinsAnimalsCell MovementCell ProliferationCells, CulturedSwinearteriovenous fistulae (AVF)arterydedifferentiationstenosisvascular smooth muscle cells (VSMCs)vein

Identifiers

PMID40243809
PMCPMC11988825

What OpenQuestion holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.