Evidence map›Paper›PMID 40191229›Full record

ArticleFrontiers in cell and developmental biology2025

Diversity of

Helen L Stott, Nir Yakoby

Abstract read
In one paragraph

Article in Frontiers in cell and developmental biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Helen L StottCenter for Computational and Integrative Biology, Rutgers, The State University of NJ, Camden, NJ, United States.
Nir YakobyCenter for Computational and Integrative Biology, Rutgers, The State University of NJ, Camden, NJ, United States.

Funding

AREA: Mechanisms underlying epithelial cells’ patterning and morphogenesisR15GM101597 · NIGMS · RUTGERS THE STATE UNIV OF NJ CAMDEN · PI YAKOBY, NIR · 2013 to 2017
$806k
NIGMS NIH HHS R15 GM101597
6 · The paper itself

Abstract

Focusing on selected model organisms to establish scientific communities and resources has greatly advanced our understanding of biological processes, including embryogenesis, and facilitated the translation of these data into developing human remedies. However, by restricting our research to a small number of model organisms, we risk overlooking the underlying mechanisms controlling animal diversity and speciation. Changes in cell signaling, protein compatibility, and genetic tinkering are often neglected due to the lack of molecular tools in non-traditional model organisms. The era of high-throughput genome sequencing, computational gene prediction, and emerging genome editing and imaging tools, offers an opportunity to explore novel mechanisms of organismal development and homeostasis. As we develop new model platforms, it is imperative to prioritize resources effectively. What criteria make an organism a "good" candidate for becoming a new model organism for exploring embryogenesis? The axis of the

Indexed as

axis formationCRISPR/cas9DrosophilaEGFR signalingmodel clade

Identifiers

PMID40191229
PMCPMC11968673

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.