ArticleCureus2025
Hypercalcemia Complicated by Rhabdomyolysis and Acute Kidney Injury.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
- Rhabdomyolysis-Induced Resistant Hypercalcemia During the Recovery Phase of Acute Kidney Injury.Cureus · 2026Article
- The Silent Surge: A Case of Rhabdomyolysis-Induced Hypercalcemia.The American journal of case reports · 2025Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Rhabdomyolysis is a condition resulting from muscle breakdown due to trauma, extreme exertion, or seizures, causing the release of intracellular contents including potassium and phosphate (Pi). Abnormalities in serum calcium (Ca) levels have also been reported in rhabdomyolysis, although its exact mechanism remains controversial. A 57-year-old male was admitted due to tonic-clonic seizures caused by epilepsy. On the second day of hospitalization, he exhibited high creatine phosphokinase (CPK) levels (39,253 U/L) and elevated serum creatinine (Cr) levels (2.31 mg/dL), indicating rhabdomyolysis and acute kidney injury (AKI). Because of anuria and azotemia, he started hemodialysis from the third day. Subsequently, renal function gradually improved, and he was weaned off hemodialysis. However, around the 20th day of hospitalization, serum Ca levels began to rise, reaching a peak of 13.3 mg/dL. A computed tomography (CT) scan showed calcification in the left-side muscles including the pectoralis major and upper arm, and bone scintigraphy also demonstrated a high accumulation of technetium-99m hydroxymethylene diphosphonate (
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Registered trials
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