Evidence map›Paper›PMID 40164668›Full record

ArticleScientific reports2025

Computational discovery of natural inhibitors targeting enterovirus D68 3C protease using molecular docking pharmacokinetics and dynamics simulations.

Mansoor Alsahag

Abstract read
In one paragraph

Article in Scientific reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

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2 · The registry

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3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

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4 · The record

Corrections and comments

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5 · Who and what money

Authors and funding

1 author.

Mansoor AlsahagFaculty of Applied Medical Sciences, Al-Baha University, Al-Baha, Kingdom of Saudi Arabia. maleshaq@bu.edu.sa.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Enterovirus D68 (EV-D68) is a significant global health threat, responsible for severe respiratory and neurological complications, with no FDA-approved antiviral treatments currently available. The 3C protease of EV-D68, an essential enzyme involved in viral replication, represents a key target for therapeutic development. In this study, we employed a comprehensive computational approach, including molecular docking, pharmacokinetic predictions, and molecular dynamics simulations, to identify potential natural inhibitors of the EV-D68 3C protease. Screening a library of natural compounds, Withaferin-A (CID: 265,237) and Baicalin (CID: 64,982) emerged as top candidates due to their favorable pharmacokinetic profiles, high binding affinities (-10.7 kcal/mol for Withaferin-A and -9.5 kcal/mol for Baicalin), and interactions with key residues in the protease's active site. The molecular dynamics simulations demonstrated the stability of the protein-ligand complexes, with low root mean square deviation (RMSD) and fluctuation (RMSF) values over a 100-ns trajectory. Free energy calculations further supported the superior binding efficiency of Withaferin-A. These findings suggest that Withaferin-A and Baicalin have significant potential as natural inhibitors of EV-D68 3C protease, offering a promising foundation for future experimental validation and the development of targeted antiviral therapies against EV-D68.

Indexed as

Antiviral AgentsCysteine EndopeptidasesEnterovirus D, HumanMolecular Docking SimulationProtease InhibitorsViral Proteins3C Viral ProteasesDrug DiscoveryEnterovirus InfectionsFlavonoidsHumansMolecular Dynamics SimulationWithanolides3C Viral ProteasesAntiviral AgentsbaicalinCysteine EndopeptidasesFlavonoidsProtease InhibitorsViral Proteinswithaferin AWithanolides3C proteaseAntiviral therapyBaicalinDynamics simulationsEnterovirus D68EV-D68Molecular modellingNatural inhibitorsPharmacokineticsWithaferin-A

Identifiers

PMID40164668
PMCPMC11958634

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