ArticleTurkish journal of surgery2025
Intergluteal fold depth has no influence on pilonidal sinus disease development.
Article in Turkish journal of surgery, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
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Who cites it
7 citing papers in PubMed.
- Advancing pilonidal sinus care through structured international collaboration: The PiloNERDs.Turkish journal of surgery · 2026Article
- Does laser treatment affect outcome in pilonidal sinus disease? Long-term multicenter retrospective analysis of pit-picking alone vs. pit-picking with laser.Techniques in coloproctology · 2026Article
- Pilonidal sinus disease and intergluteal fold depth.Turkish journal of surgery · 2025Article
- Etiologic factors in developing sacrococcygeal pilonidal sinus disease in males; A cased-control study.Langenbeck's archives of surgery · 2025Article
- Microbiome, mechanics, and morphology: Rethinking the etiopathogenesis of pilonidal sinus disease.Turkish journal of surgery · 2025Article
- Risk factors for sacrococcygeal pilonidal sinus: a systematic review and meta-analysis supplemented by genetic causal assessment.Frontiers in surgery · 2025Review
- Comparison of Phenol Application and Endoscopic Pilonidal Sinus Treatment in Pediatric Pilonidal Sinus Disease.Pediatrics international : official journal of the Japan Pediatric SocietyArticle
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Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Objective: The etiology of primary pilonidal sinus disease (PSD) remains unclear. Prior investigations suggest that sharp fragments from the occiput contribute to the formation of PSD. In 2009 a correlation between PSD and a deeper natal cleft was reported. We investigated the association between intergluteal fold (IGF) depth and PSD risk using a standardized five-step measuring protocol. Material and Methods: Our clinical prospective study included 95 PSD patients and 105 non-PSD individuals, and measurements were taken from the glabella sacralis to the anus in a northern German population. Results: The mean (± standard deviation) intergluteal depth progressively increased from the intergluteal opening from the sacral glabella at 9.1 (±3.4) mm to a maximum of 62.6 (±10.4) mm. Notably, the deepest point was consistently observed at the anus, where PSD occurrence is rare. No significant difference in IGF depth between PSD and non-PSD patients was found. Additionally, PSD predominantly developed in the proximal (cranial) third of the IGF, despite the maximum depth being in the distal region. Conclusion: These findings suggest that IGF depth is not a risk factor for PSD.
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