Evidence map›Paper›PMID 40132981›Full record

ArticleThrombosis and haemostasis2026

Homophilic Interactions of Platelet F11R/JAM-A with its Surface-Bound Counterpart Facilitate Thrombus Formation.

Piotr Kamola, Boguslawa Luzak, Patrycja Przygodzka, Cezary Watala, Moro O Salifu, Anna Babinska, Tomasz Przygodzki

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Thrombosis and haemostasis, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Piotr KamolaDepartment of Haemostatic Disorders, Medical University of Lodz, Lodz, Poland.
Boguslawa LuzakDepartment of Haemostatic Disorders, Medical University of Lodz, Lodz, Poland.
Patrycja PrzygodzkaLaboratory of Cellular and Molecular Biology, Institute of Medical Biology, Polish Academy of Sciences, Lodz, Poland.
Cezary WatalaDepartment of Haemostatic Disorders, Medical University of Lodz, Lodz, Poland.
Moro O SalifuDepartment of Medicine, State University of New York, Downstate Medical Center, Brooklyn, New York, United States.
Anna BabinskaDepartment of Medicine, State University of New York, Downstate Medical Center, Brooklyn, New York, United States.
Tomasz PrzygodzkiDepartment of Haemostatic Disorders, Medical University of Lodz, Lodz, Poland.

Funding

Narodowe Centrum Nauki UMO-2020/37/B/NZ3/00301National Science Centre grant OPUS UMO-2020/37/B/NZ3/00301
6 · The paper itself

Abstract

F11Receptor/junctional adhesion molecule-A (F11R/JAM-A) is a transmembrane protein expressed in endothelial cells, epithelial cells, and in blood platelets. In blood platelets, F11R/JAM-A participates in adhesion under static conditions, suppresses the activation of the platelet α

Indexed as

Blood PlateletsCell Adhesion MoleculesPlatelet AdhesivenessReceptors, Cell SurfaceThrombosisAnimalsAntibodies, MonoclonalCarotid Artery InjuriesDisease Models, AnimalFibrinogenHumansMaleMiceMice, Inbred C57BLPlatelet AggregationPlatelet Glycoprotein GPIIb-IIIa ComplexAntibodies, MonoclonalCell Adhesion MoleculesF11R protein, humanFibrinogenPlatelet Glycoprotein GPIIb-IIIa ComplexReceptors, Cell SurfaceRecombinant Proteins

Identifiers

PMID40132981
PMCPMC12858303

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.