Evidence map›Paper›PMID 40128102›Full record

ArticleSynapse (New York, N.Y.)2025

The Selective 5HT

Yang Wang, Jesper L Kristensen, Kristi A Kohlmeier

Abstract read
In one paragraph

Article in Synapse (New York, N.Y.), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Yang WangDepartment of Drug Design and Pharmacology, University of Copenhagen, Copenhagen, Denmark.
Jesper L KristensenDepartment of Drug Design and Pharmacology, University of Copenhagen, Copenhagen, Denmark.
Kristi A KohlmeierDepartment of Drug Design and Pharmacology, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0003-0183-3816

Funding

Chinese Scholarship CouncilS. C. Van Fund
6 · The paper itself

Abstract

Psychedelic compounds have gained renewed interest due to their rapid and long-lasting therapeutic effects on stress-related disorders. While the underlying mechanisms of therapeutic actions of psychedelic compounds are still unclear, these drugs are thought to modulate the activity of the serotonergic system, primarily through activating serotonin type 2A receptor (5-HT

Indexed as

Excitatory Postsynaptic PotentialsHallucinogensNeuronsPrefrontal CortexPyramidal CellsReceptor, Serotonin, 5-HT2ASerotonin 5-HT2 Receptor AgonistsAnimalsMaleMiceMice, Inbred C57BLPatch-Clamp TechniquesHallucinogensReceptor, Serotonin, 5-HT2ASerotonin 5-HT2 Receptor Agonistsaction potentialbrain sliceselectrophysiological recordingspsychedelicsserotonin

Identifiers

PMID40128102
PMCPMC11932889

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.