ArticleScientific reports2025
Innovative peptide therapeutics targeting IL17RA to regulate inflammatory responses.
Article in Scientific reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
4 citing papers in PubMed.
- Anti-inflammatory effect of novel polypeptide WMX-8 through specific blockade of the IL17RA signaling pathway.Frontiers in pharmacology · 2026Article
- FromFrontiers in immunology · 2026Article
- Ferroptosis-immune-metabolic axis in asthma: mechanistic crosstalk, endotype-specific regulation, and translational targeting.Frontiers in immunology · 2025Review
- The emerging role of IL-17A across different types of radiation-induced normal tissue injuries.Therapeutic advances in medical oncology · 2025Review
Corrections and comments
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Authors and funding
8 authors.
Funding
Abstract
Interleukin-17 receptor A (IL17RA) is a critical mediator of pro-inflammatory cytokine signaling and a key immune checkpoint in autoimmune diseases. While monoclonal antibodies targeting IL17RA have demonstrated clinical efficacy, their high costs, complexity in production, and lack of oral bioavailability present significant limitations. In response to these challenges, we developed AL-8(0), a novel peptide specifically designed to inhibit the IL17A-IL17RA signaling pathway. AL-8(0) was synthesized with high purity and systematically evaluated for its binding affinity and anti-inflammatory activity. Biophysical and cellular assays confirmed the peptide's strong affinity for IL17RA and its ability to inhibit inflammatory cytokine production in IL17RA-expressing monocyte-macrophages and keratinocytes. Moreover, its anti-inflammatory effects were com-parable to IL17RA-targeting monoclonal antibodies and were dependent on IL17RA expression, as demonstrated by experiments using IL17RA-deficient cells. These results underscore AL-8(0)'s potential as a targeted therapeutic for autoimmune diseases, offering a peptide-based alternative with lower antigenicity, improved scalability, and potential for oral administration. This study lays the groundwork for further development of AL-8(0) and similar peptides as innovative treatments for inflammatory disorders driven by the IL17A-IL17RA pathway.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.