Evidence map›Paper›PMID 40108106›Full record

ArticleBiochemical genetics2026

Truncated Variants in FAM20A and WDR72 Genes Underlie Autosomal Recessive Amelogenesis Imperfecta in Four Pakistani Families.

Sadaqat Ullah, Sher Alam Khan, Samin Jan, Salah Ud Din, Nazif Muhammad, Zia Ur Rehman, Abid Jan, Muhammad Tariq, Noor Muhammad, Abdul Ghani and 2 more

Abstract read
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Article in Biochemical genetics, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

12 authors.

Sadaqat UllahDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Sher Alam KhanDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Samin JanDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Salah Ud DinDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Nazif MuhammadDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Zia Ur RehmanDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Abid JanDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Muhammad TariqDepartment of Medical Laboratory Technology, University College of Duba, University of Tabuk, Tabuk, Kingdom of Saudi Arabia.
Noor MuhammadDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Abdul GhaniDepartment of Chemistry, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan.
Naveed WasifInstitute of Human Genetics, Ulm University and Ulm University Medical Center, 89081, Ulm, Germany. naveedwasif@gmail.com.
Saadullah KhanDepartment of Biotechnology and Genetic Engineering, Kohat University of Science & Technology (KUST), Khyber Pakhtunkhwa, Kohat, Pakistan. saad@kust.edu.pk.

Funding

Higher Education Commision, Pakistan No.4857/NRPU/R&D/HEC 2014
6 · The paper itself

Abstract

Amelogenesis Imperfecta (AI) is a set of hereditary diseases affecting enamel development, leading to various types of enamel defects, potentially impacting oral health unassociated with other generalized defects. AI manifests in syndromic and non-syndromic forms and can be inherited through autosomal recessive, autosomal dominant, or X-linked inheritance patterns. Genetic studies have identified sequence variants in a number of genes (≥ 70) linked to both syndromic and non-syndromic AI, highlighting the genetic diversity underlying the condition. The current study involved clinical evaluation and exome sequencing, aimed at identifying the causative variants in four unrelated consanguineous Pakistani families presenting AI phenotypes. The exome sequencing results revealed a novel homozygous frameshift variant FAM20A: NM_017565.4, c.188dupA; p.(Asp63Glufs*17) in families A, B, and C while a nonsense homozygous variant WDR72: NM_182758.4, c.2686C > T; p. (Arg896*) in family D. The segregation of both variants was confirmed by Sanger sequencing. Bioinformatics analysis predicted the pathogenicity of these genetic variants. These alterations suggest functional consequences, potentially impairing the FAM20A and WDR72 proteins and causing dental anomalies. This investigation significantly broadens our understanding of FAM20A and WDR72's involvement in AI. Furthermore, this study highlights the genetic heterogeneity of AI (involving FAM20A and WDR72 in this study) within the Pakistani population.

Indexed as

Amelogenesis ImperfectaDental Enamel ProteinsGenes, RecessiveAdolescentChildFemaleHumansMalePakistanPedigreeDental Enamel ProteinsFAM20A protein, humanAmelogenesis imperfectaConsanguinityEnamel defectsExome sequencingFAM20ASegregation analysisWDR72

Identifiers

PMID40108106
PMCPMC12882964

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.