In one paragraphArticle in American journal of physiology. Endocrinology and metabolism, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
7 authors.
Tomás Cabeza De BacaPhoenix Epidemiology and Clinical Research Branch, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Phoenix, Arizona, United States.ORCID 0000-0003-3322-2163 Emma J StinsonPhoenix Epidemiology and Clinical Research Branch, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Phoenix, Arizona, United States.ORCID 0000-0003-4875-7048 Paolo PiaggiPhoenix Epidemiology and Clinical Research Branch, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Phoenix, Arizona, United States.ORCID 0000-0003-2774-9161 Jonathan KrakoffPhoenix Epidemiology and Clinical Research Branch, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Phoenix, Arizona, United States.
Funding
Research BaseP30DK072476 · NIDDK · LSU PENNINGTON BIOMEDICAL RESEARCH CTR · PI Peter Todd Katzmarzyk · 2005 to 2026
$26.5MCoordinating Center for CALERIEU01AG022132 · NIA · DUKE UNIVERSITY · PI KRAUS, WILLIAM E · 2002 to 2013
$24.9MFactors which predict variance in weight changeZIADK069029 · NIDDK · NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES · PI KRAKOFF, JONATHAN · 2009 to 2025
$15.0MMetabolic Adaptations to Two Year Caloric RestrictionU01AG020478 · NIA · LSU PENNINGTON BIOMEDICAL RESEARCH CTR · PI RAVUSSIN, ERIC · 2002 to 2008
$13.4MCaloric Restriction and Aging in HumansU01AG020487 · NIA · WASHINGTON UNIVERSITY · PI HOLLOSZY, JOHN O. · 2001 to 2009
$8.9MDietary Energy Restriction and Metabolic Aging in HumansU01AG020480 · NIA · TUFTS UNIVERSITY BOSTON · PI ROBERTS, SUSAN B · 2002 to 2008
$8.2MFat Cell Size, Overfeeding & Ectopic FatR01DK060412 · NIDDK · LSU PENNINGTON BIOMEDICAL RESEARCH CTR · PI RAVUSSIN, ERIC · 2001 to 2011
$5.4MThe role of mitochondrial energetics in adaptive thermogenesis with overfeedingK01DK089005 · NIDDK · LSU PENNINGTON BIOMEDICAL RESEARCH CTR · PI JOHANNSEN, DARCY LARAE · 2010 to 2014
$574kHHS | National Institutes of Health (NIH) U01AG020478HHS | National Institutes of Health (NIH) U01AG020480HHS | National Institutes of Health (NIH) U01AG020487HHS | National Institutes of Health (NIH) U01AG022132HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) DK-069029-11HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) K01-DK-089005HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) P30-DK-072476HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) R01-DK-060412Intramural NIH HHS ZIA DK069029NIA NIH HHS U01 AG020478NIA NIH HHS U01 AG020480NIA NIH HHS U01 AG020487NIA NIH HHS U01 AG022132NIDDK NIH HHS K01 DK089005NIDDK NIH HHS P30 DK072476NIDDK NIH HHS R01 DK060412
6 · The paper itselfAbstract
Leptin is a hormone produced by adipocytes that plays a crucial role in regulating energy homeostasis and body mass. Despite its close correlation with body fat, up to ∼40% of variation in plasma leptin concentration remains unexplained, allowing for the classification of a distinct "leptin phenotype." This leptin phenotype-characterized by either relatively high or relatively low leptin concentration relative to an individual's level of body fat-presents an intriguing opportunity to test whether relatively higher (compared with lower) leptin concentrations differentially affect energy expenditure, metabolic adaptation, and susceptibility to weight change in response to energy balance perturbations. To test this hypothesis, we characterized the energy expenditure and weight change response between the two leptin phenotypes (relatively high vs. low) using three distinct experimental contexts: a cross-sectional analysis (
Indexed as
Energy MetabolismLeptinStress, PhysiologicalWeight GainAdultBody WeightCaloric RestrictionCross-Sectional StudiesFastingFemaleHumansMalePhenotypeYoung AdultLeptincaloric restrictionenergy expenditureleptinmetabolic adaptationoverfeeding
Identifiers
PMID40094227
PMCPMC12067472
What OpenQuestion holds
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