ArticleJournal of Cancer2025
System Analysis Identifies MYBL2 As a Novel Oncogene Target for Metastatic Prostate Cancer.
Article in Journal of Cancer, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
3 citing papers in PubMed.
- Integrative Machine Learning and Experimental Validation Identify MYBL2 as a Prognostic Biomarker and Therapeutic Target in Hepatocellular Carcinoma.Oncology research · 2026Article
- Review
- The genetic architecture of bone metastases: unveiling the role of epigenetic and genetic modifications in drug resistance.Cancer drug resistance (Alhambra, Calif.) · 2025Review
Corrections and comments
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Bone metastasis significantly contributes to the unfavorable prognosis observed in patients with prostate cancer. MYB proto-oncogene like 2 (MYBL2) has been identified as a potential target gene implicated in tumor progression. Nevertheless, the oncogenic role and underlying mechanisms of MYBL2 in bone metastasis of prostate cancer (PCa) have yet to be elucidated. Bioinformatics analyses were employed to identify genes pivotal to metastatic PCa. Subsequently, a series of molecular biology experiments in vitro, alongside a model of PCa bone metastasis in vivo, were utilized to validate the pro-metastatic effects and underlying mechanisms of MYBL2. A bioinformatics analysis identified a candidate set of 72 genes, which was used to establish a PFS prognostic model highlighting 16 key genes. Based on the expression of these 16 key genes, 498 patients with PCa from the TCGA database were divided into four subgroups. Patients in the C1 and C4 subgroups had poorer prognoses. Through the analysis of sequencing data from the C1 and C4 cohorts in comparison to the C2 and C3 cohorts, we identified MYBL2 as a critical prognostic gene in metastatic PCa. Notably, we found that MYBL2 was significantly expressed in metastatic PCa and positively related to poor prognosis. Mechanistic studies revealed that MYBL2 overexpression promoted PCa cells invasion and EMT, while NOTCH3 knockdown partly abrogated that. Moreover, MYBL2 overexpression can promote PCa xenograft growth and bone metastasis in vivo. This study found that MYBL2 overexpression in PCa were positively related to metastasis and poor prognosis. MYBL2 promoted PCa bone metastasis via activating NOTCH3. Targeting the MYBL2/NOTCH3 axis could help prevent metastatic PCa.
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Registered trials
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