ReviewNeurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology2025
Non-nystagmus hyperkinetic eye movement disorders.
Review in Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
2 citing papers in PubMed.
- Adult-onset ocular flutter-opsoclonus spectrum across diverse neurological disorders: a video-oculographic case series.BMC neurology · 2026Article
- Functional Movement Disorder: Evolving Mechanisms, Diagnostic Precision, and Personalized Multidisciplinary Care.Movement disorders clinical practice · 2025Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundWhile nystagmus is an involuntary ocular movement relatively frequent, well described, and easily recognized by most practitioners, non-nystagmus hyperkinetic eye movement disorders (NHEMD) are less obvious and can be overlooked. NHEMD may arise from intrinsic abnormalities in ocular muscles and oculomotor nerves, brain and brainstem lesions, systemic diseases, dysimmune disorders, drugs or can even be functional in nature. Given that some treatable disorders initially manifest with NHEMD, their knowledge becomes crucial.
aimsThis review aims to offer a practical and comprehensive guide to recognize specific types of NHEMD, to choose the appropriate diagnostic tests and treatments.
resultsThe review approaches individual NHEMD based on the location of anatomical lesions and the underlying pathophysiological mechanisms; therefore, it begins with those arising from abnormalities in extraocular muscles or oculomotor nerves (as for example, superior oblique myokymia and ocular neuromyotonia), then moves to NHEMD due to brainstem and brain involvement (as for example, ocular bobbing, opsoclonus, and tonic downward gaze deviation). Lastly, functional NHEMD and nonnystagmus ocular hyperkinesias associated with specific neurological disorders, such as Parkinson's disease or blepharospasm will be considered.
conclusionOverall, the review offers a comprehensive reappraisal of hyperkinetic disorders of eye motility.
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Identifiers
40080371What OpenQuestion holds
Registered trials
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