ArticleBMJ neurology open2025
CSF oligoclonal bands in neurological diseases besides CNS inflammatory demyelinating disorders.
Article in BMJ neurology open, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
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Who cites it
8 citing papers in PubMed.
- Immune dysregulation and epileptogenesis.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2026Review
- Exploring the Type 4 Cerebrospinal Fluid Oligoclonal Band Pattern: Clinical and Paraclinical Features with Diagnostic Implications.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2026Article
- Cerebellar Involvement in Immune-Mediated Pediatric Acute Ataxia: Clinical Spectrum, MRI Findings, and Predictors of MOG Antibody Positivity.Cerebellum (London, England) · 2026Article
- Multimodal MRI links gray matter volume loss and temporal lobe susceptibility to CSF immunological profiles in Guillain-Barré syndrome.Frontiers in immunology · 2026Article
- Case report of anti-vimentin IgG-associated autoimmune encephalitis: diagnostic challenges and the role of 18F-DPA714 PET/MRI.BMJ neurology open · 2026Article
- Predicting intrathecal immunoglobulin synthesis in the ICU: a comparative study of IgG-based indexes.Annals of intensive care · 2025Article
- Review
- Differential diagnoses of MS in clinical practice: incidence and methods for differential diagnosis.Frontiers in neurology · 2025Article
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7 authors.
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Abstract
Objective: To understand the distribution characteristics and clinical significance of cerebrospinal fluid (CSF) oligoclonal bands (OCBs) in different nervous system diseases besides typical central nervous system (CNS) inflammatory demyelinating disorders. Material: A total of 2259 patients who underwent CSF examination for OCBs at Peking University First Hospital from January 2011 to December 2023 were tested. A cohort of 257 patients presenting with various types of OCBs but without CNS inflammatory demyelinating diseases was included in the analysis. Relevant medical history was collected from all patients. Results: OCBs were most common in patients with autoimmune encephalitis. OCB types II and III were most common in patients with autoimmune encephalitis and CNS infection, whereas OCB types IV and V were present in immune-mediated neuropathy mostly. The distribution of OCBs also varied among different disease subtypes. Other CSF characteristics varied between diseases with different OCBs. Conclusions: In addition to CNS inflammatory demyelinating diseases, OCBs also appear in other neurological diseases including cerebral angiopathy and neurodegenerative conditions, informing the potential immune background. Further research is still needed to determine the significance of OCBs in these diseases.
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