Evidence map›Paper›PMID 40003627›Full record

ArticleLife (Basel, Switzerland)2025

Localization of Lesions in Autoimmune Blistering Diseases Is Independent of Site-Specific Target Antigen Expression.

Tina Rastegar Lari, Louis Macias, Lara Robrahn, Hasan Onur Dikmen, Jasper Prüßmann, Charlotte Kiehne, Simon Engster, Imke Weyers, Silke Szymczak, Nina van Beek and 3 more

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Life (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Tina Rastegar LariLübeck Institute of Experimental Dermatology, University of Lübeck, 23562 Lübeck, Germany.
Louis MaciasInstitute of Medical Biometry and Statistics, University of Lübeck, 23562 Lübeck, Germany.
Lara RobrahnDepartment of Dermatology, Allergology and Venerology, University of Lübeck, 23562 Lübeck, Germany.
Hasan Onur DikmenLübeck Institute of Experimental Dermatology, University of Lübeck, 23562 Lübeck, Germany.ORCID 0000-0002-0103-3942
Jasper PrüßmannDepartment of Dermatology, Allergology and Venerology, University of Lübeck, 23562 Lübeck, Germany.ORCID 0000-0002-1289-852X
Charlotte KiehneLübeck Institute of Experimental Dermatology, University of Lübeck, 23562 Lübeck, Germany.
Simon EngsterLübeck Institute of Experimental Dermatology, University of Lübeck, 23562 Lübeck, Germany.
Imke WeyersInstitute of Anatomy, University of Lübeck, 23562 Lübeck, Germany.
Silke SzymczakInstitute of Medical Biometry and Statistics, University of Lübeck, 23562 Lübeck, Germany.ORCID 0000-0002-8897-9035
Nina van BeekDepartment of Dermatology, Allergology and Venerology, University of Lübeck, 23562 Lübeck, Germany.
Markus H HoffmannDepartment of Dermatology, Allergology and Venerology, University of Lübeck, 23562 Lübeck, Germany.
Enno SchmidtLübeck Institute of Experimental Dermatology, University of Lübeck, 23562 Lübeck, Germany.
Shirin EmtenaniLübeck Institute of Experimental Dermatology, University of Lübeck, 23562 Lübeck, Germany.ORCID 0000-0002-7965-8045

Funding

CRC 1526 Pathomechanisms of Antibody-mediated AutoimmunityCRU 303 Pemphigoid DiseasesDFG - Deutsche Forschungsgemeinschaft DFG EM 362/1-1Schleswig-Holstein Excellence Cluster Precision Medicine in Chronic Inflammation DFG EXC 2167/1, TI-3University of Lübeck J13-2022
6 · The paper itself

Abstract

Autoimmune blistering diseases (AIBDs) involve autoantibodies targeting proteins in the epidermal/epithelial desmosome (pemphigus) or basement membrane zone (pemphigoid). Despite widespread antigen distribution, lesions exhibit a scattered involvement pattern. This study maps the frequency/severity of AIBD lesions on various body parts and investigates whether differential antigen expression contributes to specific predilection sites. We analyzed affected sites presenting blisters/erosions, erythematous/urticarial lesions, and mucosal lesions in bullous pemphigoid (BP-cohort 1,

Indexed as

antigen expressionautoimmune blistering diseasebullous pemphigoidpemphiguspredilection sites

Identifiers

PMID40003627
PMCPMC11857079

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.