Evidence map›Paper›PMID 39994176›Full record

ArticleNature communications2025

Context-dependent change in the fitness effect of (in)organic phosphate antiporter glpT during Salmonella Typhimurium infection.

Noemi Santamaria de Souza, Yassine Cherrak, Thea Bill Andersen, Michel Vetsch, Manja Barthel, Sanne Kroon, Erik Bakkeren, Christopher Schubert, Philipp Christen, Patrick Kiefer and 3 more

Abstract read
In one paragraph

Article in Nature communications, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Review
  3. Journal of bacteriology · 2026
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Noemi Santamaria de SouzaDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland. sanoemi@biol.ethz.ch.ORCID http://orcid.org/0000-0001-7134-7480
Yassine CherrakDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.
Thea Bill AndersenDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.ORCID http://orcid.org/0009-0005-9725-617X
Michel VetschDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.
Manja BarthelDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.
Sanne KroonDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.ORCID http://orcid.org/0000-0002-5722-0763
Erik BakkerenSir William Dunn School of Pathology, University of Oxford, Oxford, UK.ORCID http://orcid.org/0000-0001-7970-7890
Christopher SchubertDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.ORCID http://orcid.org/0000-0002-7921-1470
Philipp ChristenDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.ORCID http://orcid.org/0000-0002-3139-2874
Patrick KieferDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.
Julia A VorholtDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.ORCID http://orcid.org/0000-0002-6011-4910
Bidong D NguyenDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland.
Wolf-Dietrich HardtDepartment of Biology, Institute of Microbiology, ETH Zürich, Zürich, Switzerland. hardt@micro.biol.ethz.ch.ORCID http://orcid.org/0000-0002-9892-6420

Funding

Deutsche Forschungsgemeinschaft (German Research Foundation) SCHU 3606/1-1European Molecular Biology Organization (EMBO) ALTF-234-2020Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (Swiss National Science Foundation) 310030_192567Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (Swiss National Science Foundation) 51NF40_180575Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (Swiss National Science Foundation) NCCR Microbiomes
6 · The paper itself

Abstract

Salmonella enterica is a frequent cause of foodborne diseases, which is attributed to its adaptability. Even within a single host, expressing a gene can be beneficial in certain infection stages but neutral or even detrimental in others as previously shown for flagellins. Mutants deficient for the conserved glycerol-3-phosphate and phosphate antiporter glpT have been shown to be positively selected in nature, clinical, and laboratory settings. This suggests that different selective pressures select for the presence or absence of GlpT in a context dependent fashion, a phenomenon known as antagonistic pleiotropy. Using mutant libraries and reporters, we investigated the fitness of glpT-deficient mutants during murine orogastric infection. While glpT-deficient mutants thrive during initial growth in the gut lumen, where GlpT's capacity to import phosphate is disadvantageous, they are counter-selected by macrophages. The dichotomy showcases the need to study the spatial and temporal heterogeneity of enteric pathogens' fitness across distinct lifestyles and niches. Insights into the differential adaptation during infection may reveal opportunities for therapeutic interventions.

Indexed as

AntiportersBacterial ProteinsSalmonella InfectionsSalmonella typhimuriumAnimalsFemaleMacrophagesMiceMice, Inbred C57BLMutationPhosphatesAntiportersBacterial ProteinsPhosphates

Identifiers

PMID39994176
PMCPMC11850910

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.