ArticleCureus2025
Rapidly Progressive Pyogenic Ventriculitis Associated With Bacterial Meningitis Caused by Streptococcus intermedius.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Ventriculitis commonly arises as a complication of various central nervous system conditions. The causes of ventriculitis include both iatrogenic conditions, such as catheter-related infections, and non-iatrogenic conditions, such as community-acquired bacterial meningitis. The incidence of pyogenic ventriculitis associated with community-acquired bacterial meningitis remains unclear. Additionally, the optimal treatment strategy for pyogenic ventriculitis secondary to community-acquired bacterial meningitis remains uncertain. A 47-year-old man presented with headache, fever, and impaired consciousness. At admission, cerebrospinal fluid analysis five days after the onset (day one) revealed elevated white blood cell count with neutrophilic predominance, increased protein levels, and significantly reduced glucose. Initial brain computed tomography (CT) showed bilateral lateral ventricular enlargement with subtle fluid accumulation in the right lateral ventricle. Despite the initiation of empirical antimicrobial therapy, follow-up CT two days later demonstrated a rapid progression of fluid accumulation in the bilateral lateral ventricles. Diffusion-weighted magnetic resonance imaging (MRI) confirmed high signal intensity within the ventricles, consistent with intraventricular pus. Emergency external ventricular drainage (EVD) was promptly performed, and cultures from the intraventricular pus identified
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