Evidence map›Paper›PMID 39943735›Full record

ReviewJournal of cellular physiology2025

Molecular Mechanisms and Therapeutic Implications of Long Non-coding RNAs in Cutaneous Biology and Disease.

Alessandro Zuccotti, Farah Al-Fatyan, Giulia D S Ferretti, Irene Bertolini, David T Long, Ozgur Sahin, Jezabel Rodriguez-Blanco, Thibaut Barnoud

Abstract readReview
In one paragraph

Review in Journal of cellular physiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Alessandro ZuccottiDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.
Farah Al-FatyanDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.
Giulia D S FerrettiDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.
Irene BertoliniMolecular and Cellular Oncogenesis Program, The Wistar Institute, Philadelphia, Pennsylvania, USA.
David T LongDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.
Ozgur SahinDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.
Jezabel Rodriguez-BlancoHollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina, USA.
Thibaut BarnoudDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.ORCID 0000-0001-5588-6281

Funding

The role of SMAD1 and SATB2 in colon patterningP20GM130457 · NIGMS · MEDICAL UNIVERSITY OF SOUTH CAROLINA · PI John J Lemasters · 2020 to 2026
$18.7M
Connecting BRCA1 functions with DNA crosslink sensitivityR35GM119512 · NIGMS · MEDICAL UNIVERSITY OF SOUTH CAROLINA · PI LONG, DAVID THOMAS · 2016 to 2025
$3.9M
Integrative Training in Oncogenic SignalingT32CA193201 · NCI · MEDICAL UNIVERSITY OF SOUTH CAROLINA · PI HOWE, PHILIP H, LONG, DAVID THOMAS · 2016 to 2025
$3.7M
Overcoming chemoresistance in triple negative breast cancerR01CA267101 · NCI · UNIVERSITY OF SOUTH CAROLINA AT COLUMBIA · PI Ozgur Sahin · 2022 to 2026
$1.8M
Inhibiting tumor growth and metastasis in highly aggressive breast cancers with centrosome amplificationR01CA251374 · NCI · UNIVERSITY OF SOUTH CAROLINA AT COLUMBIA · PI SAHIN, OZGUR · 2021 to 2025
$1.7M
Identifying Novel Therapeutics for High Risk Medulloblastoma PatientsK01NS119351 · NINDS · MEDICAL UNIVERSITY OF SOUTH CAROLINA · PI RODRIGUEZ-BLANCO, JEZABEL · 2021 to 2025
$1.1M
Genetics of Tumor Suppression by p53R00CA241367 · NCI · MEDICAL UNIVERSITY OF SOUTH CAROLINA · PI BARNOUD, THIBAUT · 2022 to 2024
$747k
NCI NIH HHS R00 CA241367NCI NIH HHS R01 CA251374NCI NIH HHS R01 CA267101NCI NIH HHS T32 CA193201NIGMS NIH HHS P20 GM130457NIGMS NIH HHS R35 GM119512NINDS NIH HHS K01 NS119351
6 · The paper itself

Abstract

Human skin is the largest organ of the human body and accounts for approximately fifteen percent of the total bodyweight. Its main physiological role is to protect the body against a wide range of environmental factors including pathogens, ultraviolet light, and injury. Importantly, the skin can regenerate and heal upon injury in large part by the differentiation of keratinocytes. Not surprisingly, dysregulation of cutaneous differentiation and self-renewal can result in a variety of skin-related pathologies, including autoimmune disease and cancer. Increasing evidence supports the premise that long non-coding RNAs (lncRNAs) act as critical mediators of gene expression and regulate important biological processes within the skin. Notably, dysregulation of lncRNAs has been shown to influence diverse physiological and pathological consequences. More recently, numerous reports have revealed new mechanistic insight on the role that lncRNAs play in skin homeostasis as well as their contribution to the pathogenesis of skin-related disorders. Here, we review the biological functions of cutaneous lncRNAs and their impact on skin homeostasis. We also describe the fundamental roles of lncRNAs in the pathogenesis of skin-related disorders, including fibrotic, autoimmune, and malignant diseases. Lastly, we will highlight how a better understanding of lncRNAs at the molecular level may reveal novel therapeutic approaches for the improvement of cutaneous disorders.

Indexed as

RNA, Long NoncodingSkinSkin DiseasesAnimalsCell DifferentiationGene Expression RegulationHomeostasisHumansKeratinocytesRNA, Long Noncodingcancercutaneous biologydifferentiationlncRNApathologyskin

Identifiers

PMID39943735
PMCPMC11939017

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.