ArticleInternational journal of molecular sciences2025
Mice with Heterozygous Deletion of Exon 3 in the
Daisuke Ariyasu, Daisuke Higa, Ryo Tokudome, Takumi Yonemori, Hayate Shimada, Shinsuke Shibata, Kimi Araki
Abstract read
In one paragraphArticle in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
7 authors.
Daisuke AriyasuDivision of Developmental Genetics, Institute of Resource Development and Analysis, Kumamoto University, Honjo 860-0811, Kumamoto, Japan.
Daisuke HigaDivision of Developmental Genetics, Institute of Resource Development and Analysis, Kumamoto University, Honjo 860-0811, Kumamoto, Japan.
Ryo TokudomeDivision of Developmental Genetics, Institute of Resource Development and Analysis, Kumamoto University, Honjo 860-0811, Kumamoto, Japan.
Takumi YonemoriDivision of Developmental Genetics, Institute of Resource Development and Analysis, Kumamoto University, Honjo 860-0811, Kumamoto, Japan.
Hayate ShimadaDivision of Developmental Genetics, Institute of Resource Development and Analysis, Kumamoto University, Honjo 860-0811, Kumamoto, Japan.
Shinsuke ShibataDivision of Microscopic Anatomy, Graduate School of Medical and Dental Sciences, Niigata University, 1-757 Asahimachi-Dori, Chuo-Ku, Niigata 951-8510, Niigata, Japan.ORCID 0000-0002-1185-9043 Kimi ArakiDivision of Developmental Genetics, Institute of Resource Development and Analysis, Kumamoto University, Honjo 860-0811, Kumamoto, Japan.ORCID 0000-0002-1396-1527 Funding
Japan Society for the Promotion of Science JP21K07849Japan Society for the Promotion of Science JP22H04922
6 · The paper itselfAbstract
Isolated Growth Hormone Deficiency Type 2 (IGHD2) is caused by a heterozygous splice site variant in intron 3 of the
Indexed as
Dwarfism, PituitaryExonsGrowth HormoneReceptors, NeuropeptideReceptors, Pituitary Hormone-Regulating HormoneRNA, MessengerSequence DeletionAnimalsDisease Models, AnimalFemaleHeterozygoteHumansMaleMicePituitary GlandGrowth HormoneReceptors, NeuropeptideReceptors, Pituitary Hormone-Regulating HormoneRNA, Messengersomatotropin releasing hormone receptorendoplasmic reticulum stressgrowth hormone releasing hormone receptorisolated growth hormone deficiency type 2transcriptome analysis
Identifiers
PMID39940828
PMCPMC11817076
What OpenQuestion holds
Textmetadata
LicenceCC BY
Read underepoch 390