In one paragraphArticle in Cells, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
8 authors.
Akiyuki NishimuraDivision of Cardiocirculatory Signaling, National Institute for Physiological Sciences (NIPS), National Institutes of Natural Sciences, Okazaki 444-8787, Japan.ORCID 0000-0003-2309-3994 Kazuhiro NishiyamaDepartment of Physiology, Graduate School of Pharmaceutical Sciences, Kyushu University, Fukuoka 812-8582, Japan.
Tomoya ItoDepartment of Physiology, Graduate School of Pharmaceutical Sciences, Kyushu University, Fukuoka 812-8582, Japan.ORCID 0000-0002-1858-6009 Xinya MiDepartment of Physiology, Graduate School of Pharmaceutical Sciences, Kyushu University, Fukuoka 812-8582, Japan.
Yuri KatoDepartment of Physiology, Graduate School of Pharmaceutical Sciences, Kyushu University, Fukuoka 812-8582, Japan.ORCID 0000-0002-9426-911X Junken AokiDepartment of Health Chemistry, Graduate School of Pharmaceutical Sciences, The University of Tokyo, Tokyo 113-0033, Japan.
Motohiro NishidaDivision of Cardiocirculatory Signaling, National Institute for Physiological Sciences (NIPS), National Institutes of Natural Sciences, Okazaki 444-8787, Japan.ORCID 0000-0002-2587-5458 Funding
Japan Agency for Medical Research and Development JP22ama121038Japan Agency for Medical Research and Development JP22zf0127007Japan Science and Technology Agency JPMJCR2024Japan Science and Technology Agency JPMJFR215TJapan Science and Technology Agency JPMJMS2023Japan Society for the Promotion of Science 21H05258Japan Society for the Promotion of Science 21H05269Japan Society for the Promotion of Science 22H02772Japan Society for the Promotion of Science 22K19395Japan Society for the Promotion of Science 24K02869Japan Society for the Promotion of Science JP21H04791Japan Society for the Promotion of Science JP21H05037Japan Society for the Promotion of Science JP21H05113Japan Society for the Promotion of Science JP24K21281Naito Foundation 50636-2Smoking Research Foundation 2024G007
6 · The paper itselfAbstract
G protein-coupled receptors (GPCRs) exist in the conformational equilibrium between inactive state and active state, where the proportion of active state in the absence of a ligand determines the basal activity of GPCRs. Although many GPCRs have different basal activity, it is still unclear whether physiological stresses such as substrate stiffness affect the basal activity of GPCRs. In this study, we identified that purinergic P2Y
Indexed as
Cell Culture TechniquesReceptors, Purinergic P2AnimalsCalciumCalcium SignalingHEK293 CellsHumansLigandsCalciumLigandspurinoceptor P2Y6Receptors, Purinergic P2basal activityCa2+ oscillationGPCRspurinergic receptorsubstrate stiffness
Identifiers
PMID39937007
PMCPMC11817550
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