Evidence map›Paper›PMID 39895015›Full record

ArticlePhysiological reports2025

Myofiber structure, sarcoplasmic reticulum Ca

V Handegard, P K Lunde, M Frisk, O Seynnes, N Ørtenblad, W E Louch, G Paulsen, T Raastad

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Physiological reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Observational
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

V HandegardDepartment of Physical Performance, Norwegian School of Sport Sciences, Oslo, Norway.ORCID https://orcid.org/0009-0003-1160-7668
P K LundeInstitute for Experimental Medical Research, Oslo University Hospital and University of Oslo, Oslo, Norway.
M FriskInstitute for Experimental Medical Research, Oslo University Hospital and University of Oslo, Oslo, Norway.
O SeynnesDepartment of Physical Performance, Norwegian School of Sport Sciences, Oslo, Norway.
N ØrtenbladDepartment of Sports Science and Clinical Biomechanics, University of Southern Denmark, Odense, Denmark.
W E LouchInstitute for Experimental Medical Research, Oslo University Hospital and University of Oslo, Oslo, Norway.
G PaulsenDepartment of Physical Performance, Norwegian School of Sport Sciences, Oslo, Norway.
T RaastadDepartment of Physical Performance, Norwegian School of Sport Sciences, Oslo, Norway.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Exercise-induced muscle damage (EIMD) is characterized by a severe and prolonged decline in force-generating capacity. However, the precise cellular mechanisms underlying the observed long-lasting decline in force-generating capacity associated with EIMD are still unclear. We investigated in vivo force generation and ex vivo Ca

Indexed as

CalciumExerciseMuscle ContractionMuscle Fibers, SkeletalMuscle, SkeletalSarcoplasmic ReticulumAdultFemaleHumansMaleSarcoplasmic Reticulum Calcium-Transporting ATPasesYoung AdultCalciumSarcoplasmic Reticulum Calcium-Transporting ATPasesCa2+ sensitivitycontractile functionexercise‐induced muscle damageSR vesicle Ca2+ handlingt‐tubules

Identifiers

PMID39895015
PMCPMC11788311

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.