Evidence map›Paper›PMID 39818951›Full record

ArticlePsychological trauma : theory, research, practice and policy2025

Evaluating self-efficacy as a treatment mechanism during an intensive treatment program for posttraumatic stress disorder.

Jonathan W Murphy, Chelsea Shotwell-Tabke, Dale L Smith, Zerbrina Valdespino-Hayden, Emily Patton, Sarah Pridgen, Philip Held

Abstract read
In one paragraph

Article in Psychological trauma : theory, research, practice and policy, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Jonathan W MurphyDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.ORCID 0000-0003-4901-2865
Chelsea Shotwell-TabkeDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.
Dale L SmithDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.
Zerbrina Valdespino-HaydenDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.
Emily PattonDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.ORCID 0000-0002-7472-5878
Sarah PridgenDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.
Philip HeldDepartment of Psychiatry and Behavioral Sciences, Rush University Medical Center.

Funding

Alcohol Prevention Research on Violence, Evidence-Based Programs, and Novel Techniques (A-PREVENT) Training ProgramT32AA031818 · NIAAA · GEORGIA STATE UNIVERSITY · PI Amanda Katherine Gilmore, LAURA F SALAZAR · 2024 to 2026
$656k
Crown Family FoundationNIAAA NIH HHS T32 AA031818United Services Automobile Association/Face the FightUS Department of DefenseWounded Warrior Project
6 · The paper itself

Abstract

objectiveAlthough traumatic exposures are common, only a small percentage of people exposed to trauma go on to develop posttraumatic stress disorder (PTSD). This phenomenon suggests that there may be psychological factors that influence posttraumatic recovery trajectories. Beliefs about one's ability to cope with traumatic events have been proposed as a mechanism of posttraumatic recovery. The present study evaluated coping self-efficacy (CSE) as a treatment mechanism.

methodData were collected from 423 military service members and veterans who completed a 2-week cognitive processing therapy-based intensive treatment program for PTSD. Linear mixed-effects models were used to evaluate the associations between CSE and clinical symptoms over time. CSE and clinical symptoms were assessed at baseline, every other day during treatment, and at posttreatment. In addition, general self-efficacy (GSE) was assessed at baseline and included in the analyses.

resultsParticipants reported that increases in CSE began early and steadily increased across all domains during treatment. In addition, decreases in PTSD and depression severity also began early and steadily decreased during treatment. Although improvements in CSE predicted decreases in clinical symptoms, changes in CSE did not precede clinical improvement. Baseline GSE was a significant predictor of clinical outcomes, but changes in clinical symptoms during treatment did not differ based on one's baseline GSE.

conclusionsThe present study demonstrated that although changes in CSE do not temporarily precede changes in clinical symptoms, changes in CSE predicted changes in clinical symptoms, suggesting that CSE may serve as an indicator of treatment response. (PsycInfo Database Record (c) 2025 APA, all rights reserved).

Indexed as

Adaptation, PsychologicalCognitive Behavioral TherapyMilitary PersonnelSelf EfficacyStress Disorders, Post-TraumaticVeteransAdultFemaleHumansMaleMiddle AgedYoung Adult

Identifiers

PMID39818951
PMCPMC13138502

What OpenQuestion holds

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LicenceCC BY-NC-ND
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.